Literature DB >> 9285293

Memory deficits and industrial toxicant exposure: a comparative study of hard metal, solvent and asbestos workers.

C M Jordan1, R D Whitman, M Harbut.   

Abstract

Memory functioning was examined in ex-factory workers with hard metal disease, resulting from exposure to alloys utilizing cobalt. Since these workers are also exposed to organic solvents and may suffer from chronic hypoxia as a result of their pulmonary disorder, solvent and asbestos workers, as well as an unexposed matched sample, served as controls. Results demonstrated deficits in the allocation of attentional resources and in short-term verbal memory. A pattern of findings across several tests suggested that repetition or delay is important for adequate memory performance in individuals exposed to hard metal, implicating a deficit in encoding or slowed consolidation.

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Year:  1997        PMID: 9285293     DOI: 10.3109/00207459709000631

Source DB:  PubMed          Journal:  Int J Neurosci        ISSN: 0020-7454            Impact factor:   2.292


  2 in total

1.  Intercellular transfer of mitochondria via tunneling nanotubes protects against cobalt nanoparticle-induced neurotoxicity and mitochondrial damage.

Authors:  Fuli Zheng; Zhousong Luo; Xinpei Lin; Wei Wang; Michael Aschner; Ping Cai; Yuan-Liang Wang; Wenya Shao; Guangxia Yu; Zhenkun Guo; Siying Wu; Huangyuan Li
Journal:  Nanotoxicology       Date:  2022-01-25       Impact factor: 5.881

2.  Effects of cobalt on membrane ATPases, oxidant, and antioxidant values in the cerebrum and cerebellum of suckling rats.

Authors:  Elmouldi Garoui; Ibtissem Ben Amara; Dorra Driss; Awatef Elwej; Semia Ellouze Chaabouni; Tahia Boudawara; Najiba Zeghal
Journal:  Biol Trace Elem Res       Date:  2013-07-16       Impact factor: 3.738

  2 in total

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