Literature DB >> 9237024

Peroxynitrite, the product of nitric oxide and superoxide, causes myocardial injury in the isolated perfused rat heart.

B L Lopez1, G L Liu, T A Christopher, X L Ma.   

Abstract

BACKGROUND: Recent studies have determined that the product of NO and superoxide is peroxynitrite (ONOO-), an anion with deleterious tissue-oxidant effects.
OBJECTIVE: To examine the effects of ONOO- on the isolated perfused rat heart. Sprague-Dawley rat hearts were perfused with a cell-free, Krebs-Henseleit solution on a Langendorf perfusion apparatus. The hearts were subjected to 30 min infusions of vehicle (control); 10 mumol/l S-nitroso-N-acetylpenicillamine (SNAP), an NO donor; 10 mumol/l pyrogallol, a superoxide generator); 10 mumol/l SNAP plus 10 mumol/l pyrogallol, a mixture that generates peroxynitrite; or 10 mumol/l SNAP plus 10 mumol/l pyrogallol plus 300 U/ml superoxide dismutase.
RESULTS: SNAP or pyrogallol alone had no effect on cardiac function at the concentration used; however, infusion of the combination of SNAP and pyrogallol resulted in significant decreases in left ventricular developed pressure (to 83 +/- 4%, P < 0.01, versus vehicle) and dp/dtmax (to 76 +/- 6.2%, P < 0.01, versus vehicle), and also resulted in a significant increase in production of lactic dehydrogenase (to 118 +/- 4%, P < 0.01, versus vehicle). The administration of superoxide dismutase with SNAP and pyrogallol reversed these deleterious effects.
CONCLUSION: These results suggest that the formation of peroxynitrite significantly enhances the toxicities of .NO and O2.- and causes marked cardiac injury.

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Year:  1997        PMID: 9237024     DOI: 10.1097/00019501-199703000-00005

Source DB:  PubMed          Journal:  Coron Artery Dis        ISSN: 0954-6928            Impact factor:   1.439


  12 in total

1.  Protein kinase C activation and cardioprotective effect of preconditioning with oxidative stress in isolated rat heart.

Authors:  A Sharma; M Singh
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2.  Biphasic effect of SIN-1 is reliant upon cardiomyocyte contractile state.

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3.  Effect of aminoguanidine on ischemia-reperfusion induced myocardial injury in rats.

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4.  Nitroxyl anion exerts redox-sensitive positive cardiac inotropy in vivo by calcitonin gene-related peptide signaling.

Authors:  N Paolocci; W F Saavedra; K M Miranda; C Martignani; T Isoda; J M Hare; M G Espey; J M Fukuto; M Feelisch; D A Wink; D A Kass
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Review 5.  Nitric oxide and peroxynitrite in health and disease.

Authors:  Pál Pacher; Joseph S Beckman; Lucas Liaudet
Journal:  Physiol Rev       Date:  2007-01       Impact factor: 37.312

6.  Peroxynitrite Increases Protein Phosphatase Activity and Promotes the Interaction of Phospholamban with Protein Phosphatase 2a in the Myocardium.

Authors:  Mark J Kohr; Jonathan P Davis; Mark T Ziolo
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7.  Ischemia induced peroxynitrite dependent modifications of cardiomyocyte MLC1 increases its degradation by MMP-2 leading to contractile dysfunction.

Authors:  Dorota Polewicz; Virgilio J J Cadete; Adrian Doroszko; Beth E Hunter; Jolanta Sawicka; Danuta Szczesna-Cordary; Peter E Light; Grzegorz Sawicki
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8.  Targeting of phospholamban by peroxynitrite decreases beta-adrenergic stimulation in cardiomyocytes.

Authors:  Mark J Kohr; Honglan Wang; Debra G Wheeler; Murugesan Velayutham; Jay L Zweier; Mark T Ziolo
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Review 9.  Role of nitric oxide in the pathophysiology of heart failure.

Authors:  Hunter C Champion; Michel W Skaf; Joshua M Hare
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10.  Modulation of myocardial contraction by peroxynitrite.

Authors:  Mark J Kohr; Steve R Roof; Jay L Zweier; Mark T Ziolo
Journal:  Front Physiol       Date:  2012-12-12       Impact factor: 4.566

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