Literature DB >> 9236338

Zinc-bis-histidinate preserves cardiac function in a porcine model of cardioplegic arrest.

S R Powell1, R L Nelson, J Finnerty, D Alexander, G Pottanat, K Kooker, R J Schiff, J Moyse, S Teichberg, A J Tortolani.   

Abstract

BACKGROUND: We examined the ability of zinc-bis-histidinate to preserve postarrest myocardial function when added to a standard crystalloid cardioplegic solution.
METHODS: Domestic pigs (35 to 50 kg) on left-sided cardiopulmonary bypass were subjected to 90 minutes of regional ischemia followed by 60 minutes of hypothermic cardioplegic arrest induced by antegrade infusion of 20 mL/kg cold St. Thomas' #2 cardioplegic solution with or without 100 mumol/L of zinc-bis-histidinate and maintained by infusion of 10 mL/kg of the same every 20 minutes. During reperfusion function was assessed at 1 and 3 hours over increasing preloads using the right-sided bypass method.
RESULTS: At roller pump flows up to 2,000 mL/min, stroke work index-end-diastolic pressure curves were significantly (p < 0.05) higher and shifted to the left in treated hearts. In a series of pigs, echocardiography was used to determine end-diastolic and end-systolic volumes. At roller pump flows up to 3,500 mL/min, end-systolic pressure-end-systolic volume curves were significantly higher and shifted to the left in treated hearts. Left ventricular ejection fraction, fractional shortening, stroke volume, and cardiac output were significantly (p < 0.05) higher in treated hearts. Electron microscopy revealed that mitochondria in tissue not at risk appeared more swollen in control hearts.
CONCLUSIONS: The results of this study support the conclusion that zinc-bis-histidinate is effective as a myocardial preservative when added to a crystalloid cardioplegic solution.

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Year:  1997        PMID: 9236338     DOI: 10.1016/s0003-4975(97)00300-7

Source DB:  PubMed          Journal:  Ann Thorac Surg        ISSN: 0003-4975            Impact factor:   4.330


  3 in total

1.  Identifying cellular mechanisms of zinc-induced relaxation in isolated cardiomyocytes.

Authors:  Ting Yi; Jonathan S Vick; Marc J H Vecchio; Kelly J Begin; Stephen P Bell; Rona J Delay; Bradley M Palmer
Journal:  Am J Physiol Heart Circ Physiol       Date:  2013-06-28       Impact factor: 4.733

2.  Zinc-induced cardiomyocyte relaxation in a rat model of hyperglycemia is independent of myosin isoform.

Authors:  Ting Yi; Yaser Cheema; Sarah M Tremble; Stephen P Bell; Zengyi Chen; Meenakumari Subramanian; Martin M LeWinter; Peter VanBuren; Bradley M Palmer
Journal:  Cardiovasc Diabetol       Date:  2012-11-02       Impact factor: 9.951

Review 3.  Critical Role of Zinc as Either an Antioxidant or a Prooxidant in Cellular Systems.

Authors:  Sung Ryul Lee
Journal:  Oxid Med Cell Longev       Date:  2018-03-20       Impact factor: 6.543

  3 in total

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