Literature DB >> 9214504

Hippocampal GABA(A) channel conductance increased by diazepam.

M Eghbali1, J P Curmi, B Birnir, P W Gage.   

Abstract

Benzodiazepines, which are widely used clinically for relief of anxiety and for sedation, are thought to enhance synaptic inhibition in the central nervous system by increasing the open probability of chloride channels activated by the inhibitory neurotransmitter gamma-aminobutyric acid (GABA). Here we show that the benzodiazepine diazepam can also increase the conductance of GABAA channels activated by low concentrations of GABA (0.5 or 5 microM) in rat cultured hippocampal neurons. Before exposure to diazepam, chloride channels activated by GABA had conductances of 8 to 53pS. Diazepam caused a concentration-dependent and reversible increase in the conductance of these channels towards a maximum conductance of 70-80 pS and the effect was as great as 7-fold in channels of lowest initial conductance. Increasing the conductance of GABAA channels tonically activated by low ambient concentrations of GABA in the extracellular environment may be an important way in which these drugs depress excitation in the central nervous system. That any drug has such a large effect on single channel conductance has not been reported previously and has implications for models of channel structure and conductance.

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Year:  1997        PMID: 9214504     DOI: 10.1038/40404

Source DB:  PubMed          Journal:  Nature        ISSN: 0028-0836            Impact factor:   49.962


  48 in total

1.  Differential regulation of synaptic GABAA receptors by cAMP-dependent protein kinase in mouse cerebellar and olfactory bulb neurones.

Authors:  Z Nusser; W Sieghart; I Mody
Journal:  J Physiol       Date:  1999-12-01       Impact factor: 5.182

2.  Effects of halothane on GABA(A) receptor kinetics: evidence for slowed agonist unbinding.

Authors:  X Li; R A Pearce
Journal:  J Neurosci       Date:  2000-02-01       Impact factor: 6.167

3.  Single-channel properties of synaptic and extrasynaptic GABAA receptors suggest differential targeting of receptor subtypes.

Authors:  S G Brickley; S G Cull-Candy; M Farrant
Journal:  J Neurosci       Date:  1999-04-15       Impact factor: 6.167

4.  Incremental conductance levels of GABAA receptors in dopaminergic neurones of the rat substantia nigra pars compacta.

Authors:  A Guyon; S Laurent; D Paupardin-Tritsch; J Rossier; D Eugène
Journal:  J Physiol       Date:  1999-05-01       Impact factor: 5.182

5.  Single-channel properties of neuronal GABAA receptors from mice lacking the 2 subunit.

Authors:  M Lorez; D Benke; B Luscher; H Mohler; J A Benson
Journal:  J Physiol       Date:  2000-08-15       Impact factor: 5.182

6.  Quantitative analysis of aspartate receptor signaling complex reveals that the homogeneous two-state model is inadequate: development of a heterogeneous two-state model.

Authors:  Joshua A Bornhorst; Joseph J Falke
Journal:  J Mol Biol       Date:  2003-03-07       Impact factor: 5.469

7.  Activation of single heteromeric GABA(A) receptor ion channels by full and partial agonists.

Authors:  Martin Mortensen; Uffe Kristiansen; Bjarke Ebert; Bente Frølund; Povl Krogsgaard-Larsen; Trevor G Smart
Journal:  J Physiol       Date:  2004-02-27       Impact factor: 5.182

8.  Antiepileptic drugs induce subcritical dynamics in human cortical networks.

Authors:  Christian Meisel
Journal:  Proc Natl Acad Sci U S A       Date:  2020-05-01       Impact factor: 11.205

9.  Increased anxiety and altered responses to anxiolytics in mice deficient in the 65-kDa isoform of glutamic acid decarboxylase.

Authors:  S F Kash; L H Tecott; C Hodge; S Baekkeskov
Journal:  Proc Natl Acad Sci U S A       Date:  1999-02-16       Impact factor: 11.205

10.  Biophysical Modeling Suggests Optimal Drug Combinations for Improving the Efficacy of GABA Agonists after Traumatic Brain Injuries.

Authors:  Shyam Kumar Sudhakar; Thomas J Choi; Omar J Ahmed
Journal:  J Neurotrauma       Date:  2019-01-08       Impact factor: 5.269

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