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Literature DB >> 9184324

Development of sidedness of asymmetric body structures in vertebrates.

M Fujinaga¹.

Abstract

Mesh：

Year: 1997 PMID： 9184324

Source DB: PubMed Journal: Int J Dev Biol ISSN： 0214-6282 Impact factor: 2.203

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9 in total

1. Determination of left/right asymmetric expression of nodal by a left side-specific enhancer with sequence similarity to a lefty-2 enhancer.

Authors: H Adachi; Y Saijoh; K Mochida; S Ohishi; H Hashiguchi; A Hirao; H Hamada
Journal: Genes Dev Date: 1999-06-15 Impact factor: 11.361

2. Targeted mutagenesis of the Hira gene results in gastrulation defects and patterning abnormalities of mesoendodermal derivatives prior to early embryonic lethality.

Authors: Catherine Roberts; Helen F Sutherland; Hannah Farmer; Wendy Kimber; Stephanie Halford; Alisoun Carey; Joshua M Brickman; Anthony Wynshaw-Boris; Peter J Scambler
Journal: Mol Cell Biol Date: 2002-04 Impact factor: 4.272

3. Multiple left-right asymmetry defects in Shh(-/-) mutant mice unveil a convergence of the shh and retinoic acid pathways in the control of Lefty-1.

Authors: T Tsukui; J Capdevila; K Tamura; P Ruiz-Lozano; C Rodriguez-Esteban; S Yonei-Tamura; J Magallón; R A Chandraratna; K Chien; B Blumberg; R M Evans; J C Belmonte
Journal: Proc Natl Acad Sci U S A Date: 1999-09-28 Impact factor: 11.205

Development of sidedness of asymmetric body structures in vertebrates.

1. Determination of left/right asymmetric expression of nodal by a left side-specific enhancer with sequence similarity to a lefty-2 enhancer.

2. Targeted mutagenesis of the Hira gene results in gastrulation defects and patterning abnormalities of mesoendodermal derivatives prior to early embryonic lethality.

3. Multiple left-right asymmetry defects in Shh(-/-) mutant mice unveil a convergence of the shh and retinoic acid pathways in the control of Lefty-1.

4. Mutations in ZIC3 and ACVR2B are a common cause of heterotaxy and associated cardiovascular anomalies.

Review 5. Establishment of left-right asymmetry in vertebrate development: the node in mouse embryos.

6. The activity of the Nodal antagonist Cerl-2 in the mouse node is required for correct L/R body axis.

7. A protein disulfide isomerase expressed in the embryonic midline is required for left/right asymmetries.

8. Split cord malformation and situs inversus totalis: case report and review of the literature.

9. Split cord malformation and situs inversus totalis: case report and review of the literature.