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Literature DB >> 9170393

Dystrophinopathy, the expanding phenotype. Dystrophin abnormalities in X-linked dilated cardiomyopathy.

A H Beggs.

Abstract

Entities: Disease Gene Mutation

Mesh：

Substances：
Dystrophin

Year: 1997 PMID： 9170393 DOI： 10.1161/01.cir.95.10.2344

Source DB: PubMed Journal: Circulation ISSN： 0009-7322 Impact factor: 29.690

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8 in total

1. Nε-lysine acetylation determines dissociation from GAP junctions and lateralization of connexin 43 in normal and dystrophic heart.

Authors: Claudia Colussi; Jessica Rosati; Stefania Straino; Francesco Spallotta; Roberta Berni; Donatella Stilli; Stefano Rossi; Ezio Musso; Emilio Macchi; Antonello Mai; Gianluca Sbardella; Sabrina Castellano; Cristina Chimenti; Andrea Frustaci; Angela Nebbioso; Lucia Altucci; Maurizio C Capogrossi; Carlo Gaetano
Journal: Proc Natl Acad Sci U S A Date: 2011-01-31 Impact factor: 11.205

2. Osteopontin-stimulated expression of matrix metalloproteinase-9 causes cardiomyopathy in the mdx model of Duchenne muscular dystrophy.

Authors: Saurabh Dahiya; Srikanth Givvimani; Shephali Bhatnagar; Natia Qipshidze; Suresh C Tyagi; Ashok Kumar
Journal: J Immunol Date: 2011-08-01 Impact factor: 5.422

Dystrophinopathy, the expanding phenotype. Dystrophin abnormalities in X-linked dilated cardiomyopathy.

1. Nε-lysine acetylation determines dissociation from GAP junctions and lateralization of connexin 43 in normal and dystrophic heart.

2. Osteopontin-stimulated expression of matrix metalloproteinase-9 causes cardiomyopathy in the mdx model of Duchenne muscular dystrophy.

Review 3. Intricacies of cardiac damage in coxsackievirus B3 infection: implications for therapy.

4. Dystrophin and the cardiomyocyte membrane cytoskeleton in the healthy and failing heart.

5. Hypokalemia-induced cardiac arrest in a Duchenne muscular dystrophy patient.

6. Possible molecular mechanisms underlying age-related cardiomyocyte apoptosis in the F344XBN rat heart.

7. Dystrophin disruption in enterovirus-induced myocarditis and dilated cardiomyopathy: from bench to bedside.

Review 8. Dystrophic Cardiomyopathy: Complex Pathobiological Processes to Generate Clinical Phenotype.