Literature DB >> 9153555

Selective activation of the JNK/AP-1 pathway in Fas-mediated apoptosis of rheumatoid arthritis synoviocytes.

K Okamoto1, K Fujisawa, T Hasunuma, T Kobata, T Sumida, K Nishioka.   

Abstract

OBJECTIVE: To evaluate the Fas-dependent signaling pathway, we examined the involvement of protein tyrosine phosphorylation and the DNA binding activity of AP-1 in rheumatoid arthritis (RA) cultured synovial cells.
METHODS: The number of dead cells was counted after treatment with anti-Fas antibody in the presence of protein tyrosine kinase or phosphatase inhibitor. Protein tyrosine phosphorylation in synoviocytes after Fas ligation was examined by immunoblot and immunoprecipitation analyses. The DNA binding activity of AP-1 was examined by electrophoretic mobility shift assay.
RESULTS: Treatment with the protein tyrosine phosphatase inhibitor, orthovanadate, significantly enhanced the apoptosis of RA synoviocytes after Fas ligation. Ligation of the Fas molecule on RA synoviocytes induced a rapid tyrosine phosphorylation of JNK (c-Jun amino-terminal kinase) and formation of the AP-1 transcription factor.
CONCLUSION: Our results strongly suggest that the JNK/AP-1 signaling pathway is activated during the process of Fas-mediated apoptosis of RA synovial cells.

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Year:  1997        PMID: 9153555     DOI: 10.1002/art.1780400521

Source DB:  PubMed          Journal:  Arthritis Rheum        ISSN: 0004-3591


  7 in total

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Authors:  T Hasunuma; T Kato; T Kobata; K Nishioka
Journal:  Springer Semin Immunopathol       Date:  1998

2.  AP1 regulation of proliferation and initiation of apoptosis in erythropoietin-dependent erythroid cells.

Authors:  S M Jacobs-Helber; A Wickrema; M J Birrer; S T Sawyer
Journal:  Mol Cell Biol       Date:  1998-07       Impact factor: 4.272

3.  c-Jun N-Terminal Kinase in Inflammation and Rheumatic Diseases.

Authors:  Monica Guma; Gary S Firestein
Journal:  Open Rheumatol J       Date:  2012-09-07

Review 4.  Apoptosis and rheumatoid arthritis: past, present, and future directions.

Authors:  J D Mountz; H C Hsu; Y Matsuki; H G Zhang
Journal:  Curr Rheumatol Rep       Date:  2001-02       Impact factor: 4.686

5.  Susceptibility of rheumatoid arthritis synovial fibroblasts to FasL- and TRAIL-induced apoptosis is cell cycle-dependent.

Authors:  Noreen Pundt; Marvin A Peters; Christina Wunrau; Simon Strietholt; Carsten Fehrmann; Katja Neugebauer; Christine Seyfert; Frans van Valen; Thomas Pap; Ingmar Meinecke
Journal:  Arthritis Res Ther       Date:  2009-02-05       Impact factor: 5.156

6.  Soluble Fas ligand inhibits angiogenesis in rheumatoid arthritis.

Authors:  Wan-Uk Kim; Seung-Ki Kwok; Kyung-Hee Hong; Seung-Ah Yoo; Jin-Sun Kong; Jongseon Choe; Chul-Soo Cho
Journal:  Arthritis Res Ther       Date:  2007       Impact factor: 5.156

7.  The Role of BACH2 in T Cells in Experimental Malaria Caused by Plasmodium chabaudi chabaudi AS.

Authors:  Chelsea L Edwards; Marcela Montes de Oca; Fabian de Labastida Rivera; Rajiv Kumar; Susanna S Ng; Yulin Wang; Fiona H Amante; Kohei Kometani; Tomohiro Kurosaki; Tom Sidwell; Axel Kallies; Christian R Engwerda
Journal:  Front Immunol       Date:  2018-11-06       Impact factor: 7.561

  7 in total

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