Literature DB >> 9099824

Expression of nitric oxide synthase in the cerebral microvasculature after traumatic brain injury in the rat.

C S Cobbs1, A Fenoy, D S Bredt, L J Noble.   

Abstract

Reduced nicotinamide adenine dinucleotide phosphate-diaphorase (NADPHd) histochemistry and nitric oxide synthase (NOS) immunocytochemistry were performed on sections of brain after moderate traumatic brain injury. There was a pronounced increase in NADPHd reactivity and an induction of the endothelial NOS (eNOS) isoform in microvessels surrounding the cortical contusion by 24 h post-injury. This altered microvascular state may contribute to barrier breakdown and hyperemia which characterize traumatic brain injury.

Entities:  

Mesh:

Substances:

Year:  1997        PMID: 9099824     DOI: 10.1016/s0006-8993(96)01429-1

Source DB:  PubMed          Journal:  Brain Res        ISSN: 0006-8993            Impact factor:   3.252


  15 in total

1.  Nerve injury induces a rapid efflux of nitric oxide (NO) detected with a novel NO microsensor.

Authors:  S M Kumar; D M Porterfield; K J Muller; P J Smith; C L Sahley
Journal:  J Neurosci       Date:  2001-01-01       Impact factor: 6.167

2.  Changes of nitric oxide and its relationship with clinical features, intracranial pressure and outcome in acute head injury.

Authors:  D Zhou; M Qiu; Y Guan; L Li
Journal:  J Tongji Med Univ       Date:  2000

Review 3.  Blood-brain barrier breakdown and neovascularization processes after stroke and traumatic brain injury.

Authors:  Roshini Prakash; S Thomas Carmichael
Journal:  Curr Opin Neurol       Date:  2015-12       Impact factor: 5.710

Review 4.  Antioxidant therapies for traumatic brain injury.

Authors:  Edward D Hall; Radhika A Vaishnav; Ayman G Mustafa
Journal:  Neurotherapeutics       Date:  2010-01       Impact factor: 7.620

Review 5.  Antioxidant therapies in traumatic brain and spinal cord injury.

Authors:  Mona Bains; Edward D Hall
Journal:  Biochim Biophys Acta       Date:  2011-11-04

6.  Human cytomegalovirus US28 found in glioblastoma promotes an invasive and angiogenic phenotype.

Authors:  Liliana Soroceanu; Lisa Matlaf; Vladimir Bezrookove; Loui Harkins; Roxanne Martinez; Mary Greene; Patricia Soteropoulos; Charles S Cobbs
Journal:  Cancer Res       Date:  2011-09-07       Impact factor: 12.701

7.  Inhibition of Inducible Nitric Oxide Synthase Attenuates Deficits in Synaptic Plasticity and Brain Functions Following Traumatic Brain Injury.

Authors:  Bo Wang; Shuangshuang Han
Journal:  Cerebellum       Date:  2018-08       Impact factor: 3.847

Review 8.  Pathogenesis of malaria and clinically similar conditions.

Authors:  Ian A Clark; Lisa M Alleva; Alison C Mills; William B Cowden
Journal:  Clin Microbiol Rev       Date:  2004-07       Impact factor: 26.132

9.  Traumatic brain injury in vivo and in vitro contributes to cerebral vascular dysfunction through impaired gap junction communication between vascular smooth muscle cells.

Authors:  Guang-Xiang Yu; Martin Mueller; Bridget E Hawkins; Babu P Mathew; Margaret A Parsley; Leoncio A Vergara; Helen L Hellmich; Donald S Prough; Douglas S Dewitt
Journal:  J Neurotrauma       Date:  2014-01-31       Impact factor: 5.269

10.  Temporal relationship of peroxynitrite-induced oxidative damage, calpain-mediated cytoskeletal degradation and neurodegeneration after traumatic brain injury.

Authors:  Ying Deng; Brian M Thompson; Xiang Gao; Edward D Hall
Journal:  Exp Neurol       Date:  2007-02-03       Impact factor: 5.330

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.