Literature DB >> 905658

The clinical disease and patterns of excretion associated with three different strains of feline caliciviruses.

R C Wardley, R C Povey.   

Abstract

Three groups of cats were infected intranasally with three different feline calicivirus strains: A3, 68/40 and M8. Each strain produced a uniformly muld upper respiratory tract disease, with glossal ulceration being the most prominent clinical sign. Virus was most consistently isolated from the oro-pharyngeal region and, in non-euthanised animals, excretion continued long after clinical signs had disappeared. It is suggested that an asymptomatic phase of excretion may be a normal sequel to FCV infections.

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Year:  1977        PMID: 905658

Source DB:  PubMed          Journal:  Res Vet Sci        ISSN: 0034-5288            Impact factor:   2.534


  18 in total

1.  Antigenic change in feline calicivirus during persistent infection.

Authors:  R P Johnson
Journal:  Can J Vet Res       Date:  1992-10       Impact factor: 1.310

2.  Evolutionary mechanisms of persistence and diversification of a calicivirus within endemically infected natural host populations.

Authors:  Karen P Coyne; Rosalind M Gaskell; Susan Dawson; Carol J Porter; Alan D Radford
Journal:  J Virol       Date:  2006-12-06       Impact factor: 5.103

Review 3.  Murine norovirus: a model system to study norovirus biology and pathogenesis.

Authors:  Christiane E Wobus; Larissa B Thackray; Herbert W Virgin
Journal:  J Virol       Date:  2006-06       Impact factor: 5.103

4.  Murine norovirus 1 infection is associated with histopathological changes in immunocompetent hosts, but clinical disease is prevented by STAT1-dependent interferon responses.

Authors:  Shannon M Mumphrey; Harish Changotra; Tara N Moore; Ellen R Heimann-Nichols; Christiane E Wobus; Michael J Reilly; Mana Moghadamfalahi; Deepti Shukla; Stephanie M Karst
Journal:  J Virol       Date:  2007-01-17       Impact factor: 5.103

5.  Genogrouping of vaccine breakdown strains (VBS) of feline calicivirus in Japan.

Authors:  K Ohe; S Sakai; T Takahasi; F Sunaga; M Murakami; A Kiuchi; M Fukuyama; K Furuhata; M Hara; Y Ishikawa; A Taneno
Journal:  Vet Res Commun       Date:  2007-05       Impact factor: 2.459

6.  FCV-VBS isolated from cats with typical symptoms caused VSD in experimental cats.

Authors:  Kyoko Ohe; Toshikazu Takahashi; Daisuke Hara; Motonobu Hara
Journal:  Vet Res Commun       Date:  2007-09-25       Impact factor: 2.459

7.  Capsid protein genetic analysis and viral spread to the spinal cord in cats experimentally infected with feline calicivirus (FCV).

Authors:  Y Fujita; Y Sato; K Ohe; S Sakai; M Fukuyama; K Furuhata; S Kishikawa; S Yamamoto; A Kiuchi; M Hara; Y Ishikawa; A Taneno
Journal:  Vet Res Commun       Date:  2005-08       Impact factor: 2.459

Review 8.  Caliciviruses. Brief review.

Authors:  M J Studdert
Journal:  Arch Virol       Date:  1978       Impact factor: 2.574

9.  Genetic characterization of feline calicivirus strains associated with varying disease manifestations during an outbreak season in Missouri (1995-1996).

Authors:  Victor G Prikhodko; Carlos Sandoval-Jaime; Eugenio J Abente; Karin Bok; Gabriel I Parra; Igor B Rogozin; Eileen N Ostlund; Kim Y Green; Stanislav V Sosnovtsev
Journal:  Virus Genes       Date:  2013-11-12       Impact factor: 2.332

10.  Transmission of feline calicivirus via the cat flea (Ctenocephalides felis).

Authors:  N Mencke; M Vobis; H Mehlhorn; J D Haese; M Rehagen; S Mangold-Gehring; U Truyen
Journal:  Parasitol Res       Date:  2009-03-10       Impact factor: 2.289

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