Literature DB >> 8996294

Angiotensin-converting enzyme inhibitor therapy affects left ventricular mass in patients with ejection fraction > 40% after acute myocardial infarction.

D B Johnson1, R E Foster, F Barilla, G G Blackwell, M Roney, A W Stanley, K Kirk, R A Orr, R J van der Geest, J H Reiber, L J Dell'Italia.   

Abstract

OBJECTIVES: We tested the hypothesis that angiotensin-converting enzyme (ACE) inhibitor therapy decreases left ventricular (LV) mass in patients with a left ventricular ejection fraction (LVEF) > 40% and no evidence of heart failure after their first acute Q wave myocardial infarction (MI).
BACKGROUND: Recently, ACE inhibitor therapy has been shown to have an early mortality benefit in unselected patients with acute MI, including patients without heart failure and a LVEF > 35%. However, the effects on LV mass and volume in this patient population have not been studied.
METHODS: Thirty-five patients with a LVEF > 40% after their first acute Q wave MI were randomized to titrated oral ramipril (n = 20) or conventional therapy (control, n = 15). Magnetic resonance imaging (MRI) performed an average of 7 days and 3 months after MI provided LV volumes and mass from summated serial short-axis slices.
RESULTS: Left ventricular end-diastolic volume index did not change in ramipril-treated patients (62 +/- 16 [SD] to 66 +/- 17 ml/m2) or in control patients (62 +/- 16 to 68 +/- 17 ml/m2), and stroke volume index increased significantly in both groups. However, LV mass index decreased in ramipril-treated patients (82 +/- 18 to 73 +/- 19 g/m2, p = 0.0002) but not in the control patients (77 +/- 15 to 79 +/- 23 g/m2). Systolic arterial pressure did not change in either group at 3-month follow-up.
CONCLUSIONS: In patients with a LVEF > 40% after acute MI, ramipril decreased LV mass, and blood pressure and LV function were unchanged after 3 months of therapy. Whether the decrease in mass represents a sustained effect that is associated with a decrease in morbid events requires further investigation.

Entities:  

Mesh:

Substances:

Year:  1997        PMID: 8996294     DOI: 10.1016/s0735-1097(96)00451-2

Source DB:  PubMed          Journal:  J Am Coll Cardiol        ISSN: 0735-1097            Impact factor:   24.094


  4 in total

1.  The effects of candesartan on left ventricular hypertrophy and function in nonobstructive hypertrophic cardiomyopathy: a pilot, randomized study.

Authors:  Martin Penicka; Pavel Gregor; Roman Kerekes; Dan Marek; Karol Curila; Jiri Krupicka
Journal:  J Mol Diagn       Date:  2008-12-12       Impact factor: 5.568

2.  Functional polymorphisms in genes of the Angiotensin and Serotonin systems and risk of hypertrophic cardiomyopathy: AT1R as a potential modifier.

Authors:  Eliecer Coto; María Palacín; María Martín; Mónica G Castro; Julián R Reguero; Cristina García; José R Berrazueta; César Morís; Blanca Morales; Francisco Ortega; Ana I Corao; Marta Díaz; Beatriz Tavira; Victoria Alvarez
Journal:  J Transl Med       Date:  2010-07-01       Impact factor: 5.531

3.  Adverse effects of ACE inhibitors in patients with chronic heart failure and/or ventricular dysfunction : meta-analysis of randomised clinical trials.

Authors:  Antònia Agustí; Sara Bonet; Josep Maria Arnau; Xavier Vidal; Joan-Ramon Laporte
Journal:  Drug Saf       Date:  2003       Impact factor: 5.606

4.  Impact of vitamin D on cardiac structure and function in chronic kidney disease patients with hypovitaminosis D: a randomized controlled trial and meta-analysis.

Authors:  Debasish Banerjee; Nihil Chitalia; Irina Chis Ster; Evan Appelbaum; Ravi Thadhani; Juan Carlos Kaski; David Goldsmith
Journal:  Eur Heart J Cardiovasc Pharmacother       Date:  2021-07-23
  4 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.