Literature DB >> 8968758

De novo mutation of GDNF, ligand for the RET/GDNFR-alpha receptor complex, in Hirschsprung disease.

S M Ivanchuk1, S M Myers, C Eng, L M Mulligan.   

Abstract

Hirschsprung disease (HSCR) is a common congenital abnormality characterized by absence of the enteric ganglia in the hind gut. In 10-40% of HSCR cases, mutations of the RET receptor tyrosine kinase have been found. The recent identification of a multimeric RET ligand/receptor complex suggested that mutations of genes encoding other components of this complex might also occur in HSCR. To investigate this role, we examined the gene for glial cell line-derived neurotrophic factor (GDNF), the circulating ligand of the RET receptor complex, for mutations in a panel of sporadic and familial HSCR. We identified GDNF sequence variants in 2/36 HSCR patients. The first of these was a conservative change which did not affect the GDNF protein sequence. The second variant was a de novo missense mutation in a family with no history of HSCR and without mutation of the RET gene. Thus, our data are consistent with a causative role for GDNF mutations in some HSCR cases.

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Year:  1996        PMID: 8968758     DOI: 10.1093/hmg/5.12.2023

Source DB:  PubMed          Journal:  Hum Mol Genet        ISSN: 0964-6906            Impact factor:   6.150


  30 in total

1.  A human model for multigenic inheritance: phenotypic expression in Hirschsprung disease requires both the RET gene and a new 9q31 locus.

Authors:  S Bolk; A Pelet; R M Hofstra; M Angrist; R Salomon; D Croaker; C H Buys; S Lyonnet; A Chakravarti
Journal:  Proc Natl Acad Sci U S A       Date:  2000-01-04       Impact factor: 11.205

2.  RET genotypes comprising specific haplotypes of polymorphic variants predispose to isolated Hirschsprung disease.

Authors:  S Borrego; A Ruiz; M E Saez; O Gimm; X Gao; M López-Alonso; A Hernández; F A Wright; G Antiñolo; C Eng
Journal:  J Med Genet       Date:  2000-08       Impact factor: 6.318

Review 3.  Hirschsprung disease, associated syndromes, and genetics: a review.

Authors:  J Amiel; S Lyonnet
Journal:  J Med Genet       Date:  2001-11       Impact factor: 6.318

4.  37/67-laminin receptor facilitates neural crest cell migration during enteric nervous system development.

Authors:  Ming Fu; Amanda J Barlow-Anacker; Korah P Kuruvilla; Gary L Bowlin; Christopher W Seidel; Paul A Trainor; Ankush Gosain
Journal:  FASEB J       Date:  2020-06-27       Impact factor: 5.191

5.  Clinical relationship between EDN-3 gene, EDNRB gene and Hirschsprung's disease.

Authors:  Xiang-Long Duan; Xian-Sheng Zhang; Guo-Wei Li
Journal:  World J Gastroenterol       Date:  2003-12       Impact factor: 5.742

Review 6.  Genetic interactions and modifier genes in Hirschsprung's disease.

Authors:  Adam S Wallace; Richard B Anderson
Journal:  World J Gastroenterol       Date:  2011-12-07       Impact factor: 5.742

7.  Genetic background impacts developmental potential of enteric neural crest-derived progenitors in the Sox10Dom model of Hirschsprung disease.

Authors:  Lauren C Walters; V Ashley Cantrell; Kevin P Weller; Jack T Mosher; E Michelle Southard-Smith
Journal:  Hum Mol Genet       Date:  2010-08-25       Impact factor: 6.150

8.  Cloning and characterization of murine glial cell-derived neurotrophic factor inducible transcription factor (MGIF).

Authors:  S Yajima; C H Lammers; S H Lee; Y Hara; K Mizuno; M M Mouradian
Journal:  J Neurosci       Date:  1997-11-15       Impact factor: 6.167

Review 9.  Genetic basis of Hirschsprung's disease.

Authors:  Paul K H Tam; Mercè Garcia-Barceló
Journal:  Pediatr Surg Int       Date:  2009-06-12       Impact factor: 1.827

Review 10.  The developmental etiology and pathogenesis of Hirschsprung disease.

Authors:  Naomi E Butler Tjaden; Paul A Trainor
Journal:  Transl Res       Date:  2013-03-22       Impact factor: 7.012

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