Literature DB >> 8941098

Hyperhomocysteinemia confers an independent increased risk of atherosclerosis in end-stage renal disease and is closely linked to plasma folate and pyridoxine concentrations.

K Robinson1, A Gupta, V Dennis, K Arheart, D Chaudhary, R Green, P Vigo, E L Mayer, J Selhub, M Kutner, D W Jacobsen.   

Abstract

BACKGROUND: A high level of total plasma homocysteine is a risk factor for atherosclerosis, which is an important cause of death in renal failure. We evaluated the role of this as a risk factor for vascular complications of end-stage renal disease. METHODS AND
RESULTS: Total fasting plasma homocysteine and other risk factors were documented in 176 dialysis patients (97 men, 79 women; mean age, 56.3 +/- 14.8 years). Folate, vitamin B12, and pyridoxal phosphate concentrations were also determined. The prevalence of high total homocysteine values was determined by comparison with a normal reference population, and the risk of associated vascular complications was estimated by multiple logistic regression. Total homocysteine concentration was higher in patients than in the normal population (26.6 +/- 1.5 versus 10.1 +/- 1.7 mumol/L; P < .01). Abnormally high concentrations (> 95th percentile for control subjects, 16.3 mumol/L) were seen in 149 patients (85%) with end-stage renal disease (P < .001). Patients with a homocysteine concentration in the upper two quintiles (> 27.8 mumol/L) had an independent odds ratio of 2.9 (CI, 1.4 to 5.8; P = .007) of vascular complications. B vitamin levels were lower in patients with vascular complications than in those without. Vitamin B6 deficiency was more frequent in patients than in the normal reference population (18% versus 2%; P < .01).
CONCLUSIONS: A high total plasma homocysteine concentration is an independent risk factor for atherosclerotic complications of end-stage renal disease. Such patients may benefit from higher doses of B vitamins than those currently recommended.

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Year:  1996        PMID: 8941098     DOI: 10.1161/01.cir.94.11.2743

Source DB:  PubMed          Journal:  Circulation        ISSN: 0009-7322            Impact factor:   29.690


  35 in total

1.  Formate can differentiate between hyperhomocysteinemia due to impaired remethylation and impaired transsulfuration.

Authors:  Simon G Lamarre; Anne M Molloy; Stacey N Reinke; Brian D Sykes; Margaret E Brosnan; John T Brosnan
Journal:  Am J Physiol Endocrinol Metab       Date:  2011-09-20       Impact factor: 4.310

Review 2.  Renal dysfunction and acceleration of coronary disease.

Authors:  M W Yerkey; S J Kernis; B A Franklin; K R Sandberg; P A McCullough
Journal:  Heart       Date:  2004-08       Impact factor: 5.994

3.  Protection of podocytes from hyperhomocysteinemia-induced injury by deletion of the gp91phox gene.

Authors:  Chun Zhang; Jun-Jun Hu; Min Xia; Krishna M Boini; Christopher A Brimson; Laura A Laperle; Pin-Lan Li
Journal:  Free Radic Biol Med       Date:  2010-01-29       Impact factor: 7.376

4.  Activation of Nod-like receptor protein 3 inflammasomes turns on podocyte injury and glomerular sclerosis in hyperhomocysteinemia.

Authors:  Chun Zhang; Krishna M Boini; Min Xia; Justine M Abais; Xiang Li; Qinglian Liu; Pin-Lan Li
Journal:  Hypertension       Date:  2012-05-29       Impact factor: 10.190

Review 5.  Hyper-homocysteinemia: a novel risk factor or a powerful marker for cardiovascular diseases? Pathogenetic and therapeutical uncertainties.

Authors:  Federico Cacciapuoti
Journal:  J Thromb Thrombolysis       Date:  2011-07       Impact factor: 2.300

6.  Carotid intima-media thickness in children with end-stage renal disease on dialysis.

Authors:  A Gheissari; M Sirous; T Hajzargarbashi; R Kelishadi; A Merrikhi; A Azhir
Journal:  Indian J Nephrol       Date:  2010-01

Review 7.  Homocysteine and hydrogen sulfide in epigenetic, metabolic and microbiota related renovascular hypertension.

Authors:  Gregory J Weber; Sathnur Pushpakumar; Suresh C Tyagi; Utpal Sen
Journal:  Pharmacol Res       Date:  2016-09-04       Impact factor: 7.658

Review 8.  [Atherosclerosis and uremia: signifance of non-traditional risk factors].

Authors:  Walter H Hörl
Journal:  Wien Klin Wochenschr       Date:  2003-04-30       Impact factor: 1.704

9.  Translational upregulation of folate receptors is mediated by homocysteine via RNA-heterogeneous nuclear ribonucleoprotein E1 interactions.

Authors:  Aśok Antony; Ying-Sheng Tang; Rehana A Khan; Mangatt P Biju; Xiangli Xiao; Qing-Jun Li; Xin-Lai Sun; Hiremagalur N Jayaram; Sally P Stabler
Journal:  J Clin Invest       Date:  2004-01       Impact factor: 14.808

10.  MTHFR A1298C polymorphism is associated with cardiovascular risk in end stage renal disease in North Indians.

Authors:  Aruna Poduri; Debabrata Mukherjee; Kamal Sud; Harbir Singh Kohli; Vinay Sakhuja; Madhu Khullar
Journal:  Mol Cell Biochem       Date:  2007-09-25       Impact factor: 3.396

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