A Kishi1, N Nao-i, A Sawada. 1. Department of Ophthalmology, Miyazaki Medical College, Japan.
Abstract
PURPOSE: We studied a case of Vogt-Koyanagi-Harada syndrome with transient shallow anterior chamber and increased intraocular pressure as initial manifestations. METHODS: We examined the patient using ultrasound biomicroscopy and speculated on the mechanisms of increased intraocular pressure. RESULTS: Ultrasound biomicroscopic examinations revealed the swollen ciliary body with anterior rotation and supraciliary space at the time of increased intraocular pressure. CONCLUSIONS: We confirmed that the swollen ciliary body with anterior rotation and supraciliary space caused transient angle-closure glaucoma in this patient with Vogt-Koyanagi-Harada syndrome.
PURPOSE: We studied a case of Vogt-Koyanagi-Harada syndrome with transient shallow anterior chamber and increased intraocular pressure as initial manifestations. METHODS: We examined the patient using ultrasound biomicroscopy and speculated on the mechanisms of increased intraocular pressure. RESULTS: Ultrasound biomicroscopic examinations revealed the swollen ciliary body with anterior rotation and supraciliary space at the time of increased intraocular pressure. CONCLUSIONS: We confirmed that the swollen ciliary body with anterior rotation and supraciliary space caused transient angle-closure glaucoma in this patient with Vogt-Koyanagi-Harada syndrome.