| Literature DB >> 8895804 |
Abstract
Deficiency of GSH (BSO model) demonstrates the need for cellular protection from endogenous ROS. The pathology of various diseases is often associated with ROS and oxidative stress. The several methods for modulating cellular GSH levels presented in this chapter offer selective tools to study mechanisms and offer potential therapy of human diseases associated with GSH deficiency and oxidative stress.Entities:
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Year: 1997 PMID: 8895804 DOI: 10.1016/s1054-3589(08)60979-5
Source DB: PubMed Journal: Adv Pharmacol ISSN: 1054-3589