Literature DB >> 8873665

Creatine kinase system in failing and nonfailing human myocardium.

L Nascimben1, J S Ingwall, P Pauletto, J Friedrich, J K Gwathmey, V Saks, A C Pessina, P D Allen.   

Abstract

BACKGROUND: The creatine kinase (CK) reaction is important for rapid resynthesis of ATP when the heart increases its work. Studies defining the CK system in human failing and nonfailing myocardium are limited and in conflict. To resolve this conflict, we measured the activities of CK and its isoenzymes and the contents of creatine and CK-B in homogenates of human myocardium. METHODS AND
RESULTS: Myocardium was sampled from 23 subjects who underwent heart transplant, 36 subjects maintained in an intensive care unit before heart harvesting, 13 accident victims, and 2 patients undergoing heart surgery. Since the characteristics of myocardium of potential organ donors differed from those of myocardium of accident victims, data are presented for three groups: failing, donor, and control. CK activity was 7.7 +/- 1.9 and 6.0 +/- 1.4 IU/mg protein in left (LV) and right (RV) ventricles of failing, 9.4 +/- 2.5 and 10.7 +/- 2 IU/mg protein in LV and RV of donor, and 11.6 +/- 2.4 IU/mg protein in LV of control hearts. CK-MM and the mitochondrial isoenzyme activities were lower in failing and donor LV, and CK-MB activity and CK-B content were higher in failing and donor hearts. Creatine contents were 64 +/- 25 and 56 +/- 18.6 nmol/mg protein in LV and RV of failing, 96 +/- 30 and 110 +/- 24 nmol/mg protein in LV and RV of donor, and 131 +/- 28 nmol/mg protein in LV of control hearts.
CONCLUSIONS: In failing and nonfailing donor human myocardium, there is a combined decrease of CK activity and creatine that may impair the ability to deliver ATP to energy-consuming systems.

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Year:  1996        PMID: 8873665     DOI: 10.1161/01.cir.94.8.1894

Source DB:  PubMed          Journal:  Circulation        ISSN: 0009-7322            Impact factor:   29.690


  84 in total

1.  Reduced activity of enzymes coupling ATP-generating with ATP-consuming processes in the failing myocardium.

Authors:  P P Dzeja; D Pucar; M M Redfield; J C Burnett; A Terzic
Journal:  Mol Cell Biochem       Date:  1999-11       Impact factor: 3.396

2.  Adenoviral gene transfer of SERCA2a improves left-ventricular function in aortic-banded rats in transition to heart failure.

Authors:  M I Miyamoto; F del Monte; U Schmidt; T S DiSalvo; Z B Kang; T Matsui; J L Guerrero; J K Gwathmey; A Rosenzweig; R J Hajjar
Journal:  Proc Natl Acad Sci U S A       Date:  2000-01-18       Impact factor: 11.205

3.  Cellular remodeling in heart failure disrupts K(ATP) channel-dependent stress tolerance.

Authors:  Denice M Hodgson; Leonid V Zingman; Garvan C Kane; Carmen Perez-Terzic; Martin Bienengraeber; Cevher Ozcan; Richard J Gumina; Darko Pucar; Fergus O'Coclain; Douglas L Mann; Alexey E Alekseev; Andre Terzic
Journal:  EMBO J       Date:  2003-04-15       Impact factor: 11.598

Review 4.  Energetics and metabolism in the failing heart: important but poorly understood.

Authors:  Aslan T Turer; Craig R Malloy; Christopher B Newgard; Mihai V Podgoreanu
Journal:  Curr Opin Clin Nutr Metab Care       Date:  2010-07       Impact factor: 4.294

5.  Intracellular calcium and the relationship to contractility in an avian model of heart failure.

Authors:  C S Kim; A J Davidoff; T M Maki; A A Doye; J K Gwathmey
Journal:  J Comp Physiol B       Date:  2000-06       Impact factor: 2.200

6.  Creatine kinase MM TaqI and methylenetetrahydrofolate reductase C677T and A1298C gene polymorphisms influence exercise-induced C-reactive protein levels.

Authors:  Ana Luisa Miranda-Vilela; Arthur K Akimoto; Graciana S Lordelo; Luiz C S Pereira; Cesar K Grisolia; Maria de Nazaré Klautau-Guimarães
Journal:  Eur J Appl Physiol       Date:  2011-04-23       Impact factor: 3.078

Review 7.  CK flux or direct ATP transfer: versatility of energy transfer pathways evidenced by NMR in the perfused heart.

Authors:  F Joubert; P Mateo; B Gillet; J C Beloeil; J L Mazet; J A Hoerter
Journal:  Mol Cell Biochem       Date:  2004 Jan-Feb       Impact factor: 3.396

8.  Increasing mitochondrial ATP synthesis with butyrate normalizes ADP and contractile function in metabolic heart disease.

Authors:  Marcello Panagia; Huamei He; Tomas Baka; David R Pimentel; Dominique Croteau; Markus M Bachschmid; James A Balschi; Wilson S Colucci; Ivan Luptak
Journal:  NMR Biomed       Date:  2020-02-17       Impact factor: 4.044

9.  On the theoretical limits of detecting cyclic changes in cardiac high-energy phosphates and creatine kinase reaction kinetics using in vivo ³¹P MRS.

Authors:  Kilian Weiss; Paul A Bottomley; Robert G Weiss
Journal:  NMR Biomed       Date:  2015-04-23       Impact factor: 4.044

10.  Creatine--a dispensable metabolite?

Authors:  Heinrich Taegtmeyer; Joanne S Ingwall
Journal:  Circ Res       Date:  2013-03-15       Impact factor: 17.367

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