Literature DB >> 8832664

Results of treatment of pituitary disease in multiple endocrine neoplasia, type I.

T O'Brien1, D S O'Riordan, H Gharib, B W Scheithauer, M J Ebersold, J A van Heerden.   

Abstract

OBJECTIVE: The aim of the present study was to examine the clinical and pathological features of pituitary disease in patients with multiple endocrine neoplasia, Type I (MEN I) and to assess the prognosis.
METHODS: Fifty-two patients with pituitary disease and MEN I were studied retrospectively. Medical records were reviewed, and all of the patients known to be alive were sent a questionnaire to ascertain current disease status.
RESULTS: In 12 patients, pituitary disease was the initial manifestation of MEN I. The most common lesion was prolactinoma, followed, in frequency, by acromegaly and nonsecretory adenoma. Thirty-four of the patients had surgical treatment at the Mayo Clinic, Rochester, MN, as primary treatment, 3 had radiotherapy, and 12 received no specific therapy. Twelve patients had adjunctive radiotherapy postoperatively. Of the 34 patients receiving surgical treatment, 33 had adenoma and 1 had adenoma and pituitary hyperplasia. Immunocytochemical examination demonstrated that many tumors showed reactivity for more than one pituitary hormone. On survival analysis, no excess pituitary-related mortality was found, either in the surgically treated group or in the group as a whole.
CONCLUSION: On the basis of this study, we conclude that pituitary disease is frequently the initial manifestation of MEN I; that adenomas, particularly prolactinomas, are the rule and hyperplasia is rare; that a significant proportion of tumors are plurihormonal; and that excess pituitary-related mortality is not a factor in patients with MEN I.

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Mesh:

Year:  1996        PMID: 8832664     DOI: 10.1097/00006123-199608000-00008

Source DB:  PubMed          Journal:  Neurosurgery        ISSN: 0148-396X            Impact factor:   4.654


  15 in total

Review 1.  Pathogenesis of prolactinomas.

Authors:  Anna Spada; Giovanna Mantovani; Andrea Lania
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Review 2.  Familial pituitary tumor syndromes.

Authors:  Marianne S Elston; Kerrie L McDonald; Roderick J Clifton-Bligh; Bruce G Robinson
Journal:  Nat Rev Endocrinol       Date:  2009-06-30       Impact factor: 43.330

Review 3.  Familial isolated pituitary adenomas: from genetics to therapy.

Authors:  Federica Guaraldi; Roberto Salvatori
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4.  Pituitary tumors in MEN1: do not be misled by borderline elevated prolactin levels.

Authors:  Alina Livshits; Jelena Kravarusic; Ellie Chuang; Mark E Molitch
Journal:  Pituitary       Date:  2016-12       Impact factor: 4.107

Review 5.  Clinical genetics of multiple endocrine neoplasias, Carney complex and related syndromes.

Authors:  C A Stratakis
Journal:  J Endocrinol Invest       Date:  2001-05       Impact factor: 4.256

Review 6.  Pituitary incidentalomas.

Authors:  George A Scangas; Edward R Laws
Journal:  Pituitary       Date:  2014-10       Impact factor: 4.107

Review 7.  Current management of prolactinomas.

Authors:  P Nomikos; M Buchfelder; R Fahlbusch
Journal:  J Neurooncol       Date:  2001-09       Impact factor: 4.130

8.  Ectopic pituitary adenoma with empty sella in the setting of MEN-1 syndrome: detection with 68Ga-DOTANOC PET/CT.

Authors:  Niraj Naswa; Chandan Jyoti Das; Punit Sharma; Sellam Karunanithi; Chandrasekhar Bal; Rakesh Kumar
Journal:  Jpn J Radiol       Date:  2012-08-28       Impact factor: 2.374

9.  Silent familial isolated pituitary adenomas: histopathological and clinical case report.

Authors:  C Villa; F Magri; P Morbini; A Falchetti; P Scagnelli; E Lovati; D Locatelli; F R Canevari; V Necchi; E Gabellieri; G Guabello; L Chiovato; E Solcia
Journal:  Endocr Pathol       Date:  2008       Impact factor: 3.943

Review 10.  Genetic and epigenetic mutations of tumor suppressive genes in sporadic pituitary adenoma.

Authors:  Yunli Zhou; Xun Zhang; Anne Klibanski
Journal:  Mol Cell Endocrinol       Date:  2013-09-11       Impact factor: 4.102

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