Literature DB >> 8814146

Differential regulation of cytokine and receptor transcript expression in human corneal and limbal fibroblasts by epidermal growth factor, transforming growth factor-alpha, platelet-derived growth factor B, and interleukin-1 beta.

D Q Li1, S C Tseng.   

Abstract

PURPOSE: To explore further the significance of three patterns of cytokine dialogues that have been characterized between human corneal and limbal epithelial cells and fibroblasts.
METHODS: Northern hybridization of the transcript expression of type I cytokine receptors (EGFR, IL-1R, and PDGFR-beta), type II cytokines (bFGF, LIF, and TGF-beta 1), and type III cytokines (HGF and KGF) by human corneal and limbal fibroblasts was conducted under the modulation of TGF-alpha, PDGF-BB, IL-1 beta, and EGF (type I cytokines). The mechanism of upregulation by IL-1 beta was studied further with respect to proto-oncogene expression and under the treatment of cycloheximide and actinomycin D.
RESULTS: Results showed that EGF upregulated LIF and HGF but downregulated KGF and M-CSF. Unlike EGF, TGF-alpha upregulated additional EGFR, PDGFR-beta, bFGF, and TGF-beta 1, suggesting that although they share the same EGFR, TGF-alpha, which is produced by epithelium, is more effective in activating fibroblasts than EGF, which is present in tears. The upregulation of PDGF-BB was similar to that of TGF-alpha, except that it further stimulated IL-8, supporting their synergistic roles in promoting wound healing. Uniquely, IL-1 beta upregulated KGF expression by limbal fibroblasts more than corneal fibroblasts and IL-8 and M-CSF expression, but it downregulated PDGFR-beta. In IL-1 beta, the upregulation of cytokines and receptors was preceded by the upregulation of c-fos, c-jun, and c-myc, and it was inhibited by actinomycin D. Its upregulation of LIF was superinduced, but the upregulation of bFGF and KGF was inhibited, and that of the rest was not affected by cycloheximide.
CONCLUSIONS: These findings suggest that epithelial cells under stress or injury (producing IL-1) might preferentially activate limbal epithelial stem cells indirectly by fibroblasts and simultaneously might promote inflammation during wound healing.

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Year:  1996        PMID: 8814146

Source DB:  PubMed          Journal:  Invest Ophthalmol Vis Sci        ISSN: 0146-0404            Impact factor:   4.799


  16 in total

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4.  Osmoprotective effects of supplemental epidermal growth factor in an ex vivo multilayered human conjunctival model under hyperosmotic stress.

Authors:  Jae-hyung Kim; Soon-Suk Kang; Eun Soon Kim; Jae Yong Kim; Myoung Joon Kim; Hungwon Tchah
Journal:  Graefes Arch Clin Exp Ophthalmol       Date:  2013-05-16       Impact factor: 3.117

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Authors:  Chun-Chen Chen; Shiow-Wen Liou; Chi-Chih Chen; Wen-Chung Chen; Fung-Rong Hu; I-Jong Wang; Shing-Jong Lin
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10.  Genome-wide meta-analysis of five Asian cohorts identifies PDGFRA as a susceptibility locus for corneal astigmatism.

Authors:  Qiao Fan; Xin Zhou; Chiea-Chuen Khor; Ching-Yu Cheng; Liang-Kee Goh; Xueling Sim; Wan-Ting Tay; Yi-Ju Li; Rick Twee-Hee Ong; Chen Suo; Belinda Cornes; Mohammad Kamran Ikram; Kee-Seng Chia; Mark Seielstad; Jianjun Liu; Eranga Vithana; Terri L Young; E-Shyong Tai; Tien-Yin Wong; Tin Aung; Yik-Ying Teo; Seang-Mei Saw
Journal:  PLoS Genet       Date:  2011-12-01       Impact factor: 5.917

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