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Literature DB >> 8796338

Histamine receptors and bioeffects on clonal parathyroid endothelial cells.

U Frediani¹, S Benvenuti, L Masi, A L Tanini, M L Brandi.

Abstract

Using a clonal line of bovine parathyroid endothelial cells (BPE-1) we defined the presence on these cells of a histamine H2 receptor and characterized its pharmacological properties. Interaction of histamine with its receptor induced an increase of cAMP accumulation in a dose- and time-dependent fashion. This effect appears unique for parathyroid endothelial cells, in fact, clonal parathyroid epithelial cells did not exhibit a similar response. No effect of histamine was observed on BPE-1 cell proliferation.

Entities: Chemical Gene Species

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Year: 1996 PMID： 8796338 DOI： 10.1007/BF03347866

Source DB: PubMed Journal: J Endocrinol Invest ISSN： 0391-4097 Impact factor: 4.256

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References

33 in total

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Histamine receptors and bioeffects on clonal parathyroid endothelial cells.

1. Secretion of tissue-type plasminogen activator and plasminogen activator inhibitor by cultured human endothelial cells: modulation by thrombin, endotoxin, and histamine.

2. Effect of cimetidine on circulating parathyroid hormone in primary hyperparathyroidism.

Review 3. Endothelial cell receptors for histamine.

4. Histamine stimulate prostacyclin synthesis in cultured human umbilical vein endothelial cells.

5. Reduction by cimetidine of serum parathyroid hormone levels in uremic patients.

6. Histamine-induced phosphoinositide metabolism in cultured human umbilical vein endothelial cells. Association with thromboxane and prostacyclin release.

7. Establishment and characterization of a clonal line of parathyroid endothelial cells.

8. Isolated human coronary arteries in response to vasoconstrictor substances.

9. Role of the endothelium in the vasodilator response of rat thoracic aorta to histamine.

10. Further evidence for the distribution and nature of histamine receptors on microvascular endothelium.