Literature DB >> 878926

Longitudinal extension of oedema in experimental spinal cord injury--evidence for two types of post-traumatic oedema.

S t Nĕmecek, R Petr, P Suba, V Rozsival, O Mĕlka.   

Abstract

As in brain, another, obviously hydrostatic, type of oedema develops in the acutely injured spinal cord of the rabbit in addition to vasogenic oedema. The authors arrived at this finding by means of fluorescent tracer studies histological examinations, and measurements of tissue water, sodium and potassium. Longitudinal spread of vasogenic oedema remained limited to the zone of the epicentre and its surroundings. Hyperhydration, which appears gradually in the remote segments and is associated with increased sodium content, has the characteristics of plasma-ultrafiltrate.

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Mesh：

Year:  1977        PMID： 878926     DOI： 10.1007/bf01401922

Source DB:  PubMed          Journal:  Acta Neurochir (Wien)        ISSN： 0001-6268            Impact factor:   2.216


  27 in total

Review 1.  The pathophysiological response to spinal cord injury. The current status of related research.

Authors:  J L Osterholm
Journal:  J Neurosurg       Date:  1974-01       Impact factor: 5.115

2.  The luxury-perfusion syndrome and its possible relation to acute metabolic acidosis localised within the brain.

Authors:  N A Lassen
Journal:  Lancet       Date:  1966-11-19       Impact factor: 79.321

3.  Chemical characteristics of traumatic spinal cord edema in cats. Effects of steroids on potassium depletion.

Authors:  M G Lewin; R R Hansebout; H M Pappius
Journal:  J Neurosurg       Date:  1974-01       Impact factor: 5.115

4.  Induction of filtration edema by extreme reduction of cerebrovascular resistance associated with hypertension.

Authors:  G Meinig; H J Reulen; A Hadjidimos; C Siemon; D Bartko; K Schürmann
Journal:  Eur Neurol       Date:  1972       Impact factor: 1.710

5.  Brain swelling caused by trauma and arterial hypertension. Hemodynamic aspects.

Authors:  W J Marshall; J L Jackson; T W Langfitt
Journal:  Arch Neurol       Date:  1969-11

6.  Studies on vascular permeability changes in experimental brain concussion. I. Distribution of circulating fluorescent indicators in brain and cervical cord after sudden mechanical loading of the brain.

Authors:  L Rinder; Y Olsson
Journal:  Acta Neuropathol       Date:  1968-09-25       Impact factor: 17.088

7.  Brain swelling produced by injury and aggravated by arterial hypertension. A light and electron microscopic study.

Authors:  H S Schutta; N F Kassell; T W Langfitt
Journal:  Brain       Date:  1968-06       Impact factor: 13.501

8.  Role of histamine in posttraumatic spinal cord hyperemia and the luxury perfusion syndrome.

Authors:  A I Kobrine; T F Doyle
Journal:  J Neurosurg       Date:  1976-01       Impact factor: 5.115

9.  Microangiographic study of experimental spinal cord injuries.

Authors:  D J Fairholm; I M Turnbull
Journal:  J Neurosurg       Date:  1971-09       Impact factor: 5.115

10.  Local spinal cord blood flow in experimental traumatic myelopathy.

Authors:  A I Kobrine; T F Doyle; A N Martins
Journal:  J Neurosurg       Date:  1975-02       Impact factor: 5.115
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  6 in total

1.  Serotonin in contused spinal cord.

Authors:  S Nĕmecek; P Suba; J Cerman
Journal:  Acta Neurochir (Wien)       Date:  1977       Impact factor: 2.216

2.  Edema formation and cellular alterations following spinal cord injury in the rat and their modification with p-chlorophenylalanine.

Authors:  H S Sharma; Y Olsson
Journal:  Acta Neuropathol       Date:  1990       Impact factor: 17.088

3.  Effects of p-chlorophenylalanine on microvascular permeability changes in spinal cord trauma. An experimental study in the rat using 131I-sodium and lanthanum tracers.

Authors:  Y Olsson; H S Sharma; C A Pettersson
Journal:  Acta Neuropathol       Date:  1990       Impact factor: 17.088

4.  Increased 5-hydroxytryptamine immunoreactivity in traumatized spinal cord. An experimental study in the rat.

Authors:  H S Sharma; J Westman; Y Olsson; O Johansson; P K Dey
Journal:  Acta Neuropathol       Date:  1990       Impact factor: 17.088

5.  Spinal cord oedema due to venous stasis.

Authors:  F C Henderson; H A Crockard; J M Stevens
Journal:  Neuroradiology       Date:  1993       Impact factor: 2.804

6.  NK1 receptor blockade is ineffective in improving outcome following a balloon compression model of spinal cord injury.

Authors:  Anna Victoria Leonard; Emma Thornton; Robert Vink
Journal:  PLoS One       Date:  2014-05-23       Impact factor: 3.240
  6 in total

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