Literature DB >> 8774853

Decreased L-arginine-nitric oxide pathway in cultured myoblasts from spontaneously hypertensive versus normotensive Wistar-Kyoto rats.

G Dubois1.   

Abstract

The hypertrophy of the arterial media exhibited by spontaneously hypertensive rats is related to the hyperproliferation of their smooth muscle cells in culture. Using two methods to assay nitric oxide synthase activity, we showed that this abnormal proliferation results from a decreased activity of the inducible NOS II and less modulation of this enzymatic activity by these cells compared to control Wistar Kyoto cells.

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Year:  1996        PMID： 8774853     DOI： 10.1016/0014-5793(96)00817-4

Source DB:  PubMed          Journal:  FEBS Lett        ISSN： 0014-5793            Impact factor:   4.124


  5 in total

1.  Chronic treatment with C-type natriuretic peptide impacts differently in the aorta of normotensive and hypertensive rats.

Authors:  Carolina Caniffi; Flavia M Cerniello; Gonzalo Bouchet; María L Sueiro; Analía Tomat; Daniel González Maglio; Jorge E Toblli; Cristina Arranz
Journal:  Pflugers Arch       Date:  2019-06-11       Impact factor: 3.657

2.  Increased activity and expression of Ca(2+)-dependent NOS in renal cortex of ANG II-infused hypertensive rats.

Authors:  S Y Chin; K N Pandey; S J Shi; H Kobori; C Moreno; L G Navar
Journal:  Am J Physiol       Date:  1999-11

3.  Docosahexaenoic acid potentiates interleukin-1beta induction of nitric oxide synthase through mechanism involving p44/42 MAPK activation in rat vascular smooth muscle cells.

Authors:  Masahiko Hirafuji; Takuji Machida; Marito Tsunoda; Atsushi Miyamoto; Masaru Minami
Journal:  Br J Pharmacol       Date:  2002-06       Impact factor: 8.739

4.  Natriuretic peptide receptor-C activation attenuates angiotensin II-induced enhanced oxidative stress and hyperproliferation of aortic vascular smooth muscle cells.

Authors:  Padma Madiraju; Ekhtear Hossain; Madhu B Anand-Srivastava
Journal:  Mol Cell Biochem       Date:  2018-02-07       Impact factor: 3.396

5.  Inhibition of overexpression of Giα proteins and nitroxidative stress contribute to sodium nitroprusside-induced attenuation of high blood pressure in SHR.

Authors:  Ekhtear Hossain; Oli Sarkar; Yuan Li; Madhu B Anand-Srivastava
Journal:  Physiol Rep       Date:  2018-03
  5 in total

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