Literature DB >> 8724552

Triggers for sudden cardiac death in the athlete.

B J Maron1.   

Abstract

Sudden death on the athletic field is usually due to underlying cardiovascular disease. Coronary artery disease is most common in older athletes, and a variety of congenital cardiovascular malformations predominate in young competitive athletes. Of these lesions, the most common in North America is hypertrophic cardiomyopathy. A variety of coronary artery anomalies are next in frequency, with the most important being anomalous origin of left main coronary artery from the anterior sinus of Valsalva.

Entities:  

Mesh:

Year:  1996        PMID: 8724552     DOI: 10.1016/s0733-8651(05)70273-3

Source DB:  PubMed          Journal:  Cardiol Clin        ISSN: 0733-8651            Impact factor:   2.213


  10 in total

Review 1.  Sudden cardiac death in the young athlete.

Authors:  M M Goble
Journal:  Indian J Pediatr       Date:  1999 Jan-Feb       Impact factor: 1.967

Review 2.  The ubiquitin-proteasome system and cardiovascular disease.

Authors:  Saul R Powell; Joerg Herrmann; Amir Lerman; Cam Patterson; Xuejun Wang
Journal:  Prog Mol Biol Transl Sci       Date:  2012       Impact factor: 3.622

3.  Atomic resolution probe for allostery in the regulatory thin filament.

Authors:  Michael R Williams; Sarah J Lehman; Jil C Tardiff; Steven D Schwartz
Journal:  Proc Natl Acad Sci U S A       Date:  2016-03-08       Impact factor: 11.205

Review 4.  Congenital anomalies of coronary arteries: role in the pathogenesis of sudden cardiac death.

Authors:  Melvin D Cheitlin; John MacGregor
Journal:  Herz       Date:  2009-06       Impact factor: 1.443

Review 5.  Sudden death in athletes: an update.

Authors:  L G Futterman; R Myerburg
Journal:  Sports Med       Date:  1998-11       Impact factor: 11.136

6.  A mouse model of myosin binding protein C human familial hypertrophic cardiomyopathy.

Authors:  Q Yang; A Sanbe; H Osinska; T E Hewett; R Klevitsky; J Robbins
Journal:  J Clin Invest       Date:  1998-10-01       Impact factor: 14.808

7.  A truncated cardiac troponin T molecule in transgenic mice suggests multiple cellular mechanisms for familial hypertrophic cardiomyopathy.

Authors:  J C Tardiff; S M Factor; B D Tompkins; T E Hewett; B M Palmer; R L Moore; S Schwartz; J Robbins; L A Leinwand
Journal:  J Clin Invest       Date:  1998-06-15       Impact factor: 14.808

Review 8.  Genetic, clinical, molecular, and pathogenic aspects of the South Asian-specific polymorphic MYBPC3Δ25bp variant.

Authors:  Mohammed Arif; Pooneh Nabavizadeh; Taejeong Song; Darshini Desai; Rohit Singh; Sholeh Bazrafshan; Mohit Kumar; Yigang Wang; Richard J Gilbert; Perundurai S Dhandapany; Richard C Becker; Evangelia G Kranias; Sakthivel Sadayappan
Journal:  Biophys Rev       Date:  2020-07-12

9.  Appetite for destruction: E3 ubiquitin-ligase protection in cardiac disease.

Authors:  Monte S Willis; Jonathan C Schisler; Cam Patterson
Journal:  Future Cardiol       Date:  2008-01

Review 10.  Percutaneous treatment of a CTO in an anomalous right coronary artery: A rupture paved the way for new insights.

Authors:  Nino Cocco; Rosalinda Madonna; Valeria Cammalleri; Giulio Cocco; Domenico De Stefano; Danilo Ricciardi; Francesco Grigioni; Gian Paolo Ussia
Journal:  Front Cardiovasc Med       Date:  2022-07-29
  10 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.