Literature DB >> 8707091

Sulindac induced regression of colorectal adenomas in familial adenomatous polyposis: evaluation of predictive factors.

F M Giardiello1, J A Offerhaus, A C Tersmette, L M Hylind, A J Krush, J D Brensinger, S V Booker, S R Hamilton.   

Abstract

BACKGROUND: Sulindac, a non-steroidal anti-inflammatory drug, causes regression of colorectal adenomas in patients with familial adenomatous polyposis (FAP) but the response is variable. Specific clinical factors predictive of sulindac induced regression have not been studied.
METHODS: 22 patients with FAP were given sulindac 150 mg orally twice a day. Polyp number and size were determined before treatment and at three months. The relation of nine clinical factors to polyp regression (per cent of baseline polyp number after treatment) was evaluated by univariate and multivariate analysis.
RESULTS: After three months of sulindac, polyp number had decreased to 45 per cent of baseline and polyp size to 50 per cent of baseline (p < 0.001 and p < 0.01, respectively). Univariate analysis showed greater polyp regression in older patients (p = 0.004), those with previous colectomy and ileorectal anastomosis (p = 0.001), and patients without identifiable mutation of the APC gene responsible for FAP (p = 0.05). With multivariate regression analysis, response to sulindac treatment was associated with previous subtotal colectomy.
CONCLUSIONS: Sulindac treatment seems effective in producing regression of colorectal adenomas of FAP patients with previous subtotal colectomy regardless of baseline polyp number and size. Changed sulindac metabolism, reduced area of the target mucosa, or changed epithelial characteristics after ileorectal anastomosis may explain these findings.

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Year:  1996        PMID: 8707091      PMCID: PMC1383118          DOI: 10.1136/gut.38.4.578

Source DB:  PubMed          Journal:  Gut        ISSN: 0017-5749            Impact factor:   23.059


  18 in total

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2.  Effect of sulindac on small polyps in familial adenomatous polyposis.

Authors:  H S Debinski; J Trojan; K P Nugent; A D Spigelman; R K Phillips
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4.  Randomized controlled trial of the effect of sulindac on duodenal and rectal polyposis and cell proliferation in patients with familial adenomatous polyposis.

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5.  Identification of FAP locus genes from chromosome 5q21.

Authors:  K W Kinzler; M C Nilbert; L K Su; B Vogelstein; T M Bryan; D B Levy; K J Smith; A C Preisinger; P Hedge; D McKechnie
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6.  Antiproliferative effect of nonsteroidal antiinflammatory drugs against human colon cancer cells.

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7.  Treatment of colonic and rectal adenomas with sulindac in familial adenomatous polyposis.

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Authors:  S M Powell; G M Petersen; A J Krush; S Booker; J Jen; F M Giardiello; S R Hamilton; B Vogelstein; K W Kinzler
Journal:  N Engl J Med       Date:  1993-12-30       Impact factor: 91.245

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7.  Dietary putrescine reduces the intestinal anticarcinogenic activity of sulindac in a murine model of familial adenomatous polyposis.

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10.  Sulindac effects on inflammation and tumorigenesis in the intestine of mice with Apc and Mlh1 mutations.

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