Literature DB >> 8670772

Propranolol suppression of ocular HSV-1 recurrence and associated corneal lesions following spontaneous reactivation in the rabbit.

H E Kaufman1, E D Varnell, B M Gebhardt, H W Thompson, J M Hill.   

Abstract

PURPOSE: Hyperthermia has been shown to induce HSV-1 ocular shedding in mice. Systemic administration of propranolol significantly reduced the recovery of infectious virus in the tears, cornea, and trigeminal ganglia of mice subjected to hyperthermia. The present study was performed to determine the effects of systemic propranolol on ocular shedding and recurrent corneal epithelial lesions in the rabbit model.
METHODS: New Zealand white rabbits were infected with HSV-1 strain McKrae or 17Syn+. After latency was established, the animals were treated with systemic propranolol or saline (control) and examined by slit lamp biomicroscopy for corneal lesion. Tear film swabs were cultured to determine the frequency and duration of viral shedding.
RESULTS: Propranolol caused a significant reduction in the frequency and duration of ocular HSV-1 shedding and a reduction in the frequency of recurrent corneal epithelia disease, compared with saline treatment.
CONCLUSIONS: These results suggest that beta-adrenergic receptor blockers such as propranolol could be useful in suppressing HSV-1 ocular recurrences and corneal disease.

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Year:  1996        PMID: 8670772     DOI: 10.3109/02713689609008909

Source DB:  PubMed          Journal:  Curr Eye Res        ISSN: 0271-3683            Impact factor:   2.424


  6 in total

Review 1.  Suppression of herpes simplex virus 1 reactivation in a mouse eye model by cyclooxygenase inhibitor, heat shock protein inhibitor, and adenosine monophosphate.

Authors:  Yoshikazu Shimomura; Shiro Higaki; Keizo Watanabe
Journal:  Jpn J Ophthalmol       Date:  2010-06-25       Impact factor: 2.447

2.  Identical 371-base-pair deletion mutations in the LAT genes of herpes simplex virus type 1 McKrae and 17syn+ result in different in vivo reactivation phenotypes.

Authors:  J M Loutsch; G C Perng; J M Hill; X Zheng; M E Marquart; T M Block; H Ghiasi; A B Nesburn; S L Wechsler
Journal:  J Virol       Date:  1999-01       Impact factor: 5.103

Review 3.  Rabbit Models for Studying Human Infectious Diseases.

Authors:  Xuwen Peng; John A Knouse; Krista M Hernon
Journal:  Comp Med       Date:  2015-12       Impact factor: 0.982

4.  HSV-1 latent rabbits shed viral DNA into their saliva.

Authors:  James M Hill; Nicole M Nolan; Harris E McFerrin; Christian Clement; Timothy P Foster; William P Halford; Konstantin G Kousoulas; Walter J Lukiw; Hilary W Thompson; Ethan M Stern; Partha S Bhattacharjee
Journal:  Virol J       Date:  2012-09-28       Impact factor: 4.099

5.  Analysis of the mechanism of reactivation of latently infecting pseudorabies virus by acetylcholine.

Authors:  Seiichi Tanaka; Kazuaki Mannen
Journal:  J Vet Med Sci       Date:  2014-01-10       Impact factor: 1.267

Review 6.  Rabbit and mouse models of HSV-1 latency, reactivation, and recurrent eye diseases.

Authors:  Jody M Webre; James M Hill; Nicole M Nolan; Christian Clement; Harris E McFerrin; Partha S Bhattacharjee; Victor Hsia; Donna M Neumann; Timothy P Foster; Walter J Lukiw; Hilary W Thompson
Journal:  J Biomed Biotechnol       Date:  2012-10-02
  6 in total

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