Literature DB >> 8655957

Quantitative analysis of hepatitis C virus RNA in liver biopsies by competitive reverse transcription and polymerase chain reaction.

G Grassi1, G Pozzato, M Moretti, M Giacca.   

Abstract

BACKGROUND/AIMS: The determination of HCV-RNA concentration in liver samples is likely to provide interesting insights for the study of disease progression and for evaluation of the efficacy of anti-viral therapy.
METHODS: A procedure was developed for the precise quantification of HCV-RNA in liver biopsies, based on the competitive reverse transcription-polymerase chain reaction technology. This competitive assay consists of the co-amplification of the target RNA with known amounts of a competitor RNA molecule containing the same sequence as the target plus an insertion in the middle, allowing resolution of the two amplification products by gel electrophoresis.
RESULTS: The amounts of HCV-genomic-RNA and beta-actin mRNA (the latter being used as an internal standard to overcome the problem of reproducibility of quantitative RNA extraction) were evaluated in liver biopsies of 15 patients affected by hepatitis C virus-positive chronic liver disease at the time of diagnosis. All the patients underwent alpha-interferon therapy for 6 months and were subsequently followed for at least 1 further year after the end of treatment. Viral RNA concentration (which ranged from 2 to 2.7 x 10(5) HCV-RNA molecules per 10(6) beta-actin molecules) directly correlated with the efficacy of treatment, indicating that low levels of viral replication in the liver are associated with a poor response to therapy.
CONCLUSIONS: This study suggests that the determination of viral load in the liver is an important prognostic tool for the prediction of the efficacy of alpha-interferon therapy.

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Year:  1995        PMID: 8655957     DOI: 10.1016/0168-8278(95)80198-7

Source DB:  PubMed          Journal:  J Hepatol        ISSN: 0168-8278            Impact factor:   25.083


  8 in total

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2.  Dynamics of HIV-1 mRNA expression in patients with long-term nonprogressive HIV-1 infection.

Authors:  M Comar; C Simonelli; S Zanussi; P Paoli; E Vaccher; U Tirelli; M Giacca
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3.  Predictors of response to interferon therapy.

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Journal:  Dig Dis Sci       Date:  1996-12       Impact factor: 3.199

4.  Selectively reduced tat mRNA heralds the decline in productive human immunodeficiency virus type 1 infection in monocyte-derived macrophages.

Authors:  Secondo Sonza; Helen P Mutimer; Kate O'Brien; Philip Ellery; Jane L Howard; Jonathan H Axelrod; Nicholas J Deacon; Suzanne M Crowe; Damian F J Purcell
Journal:  J Virol       Date:  2002-12       Impact factor: 5.103

5.  An exonic splicing silencer downstream of the 3' splice site A2 is required for efficient human immunodeficiency virus type 1 replication.

Authors:  Joshua M Madsen; C Martin Stoltzfus
Journal:  J Virol       Date:  2005-08       Impact factor: 5.103

6.  Dissociation of serum and liver hepatitis C virus RNA levels in patients coinfected with human immunodeficiency virus and treated with antiretroviral drugs.

Authors:  Milena Furione; Renato Maserati; Marta Gatti; Fausto Baldanti; Agostino Cividini; Raffaele Bruno; Giuseppe Gerna; Mario U Mondelli
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7.  Human immunodeficiency virus type 1 infection inhibits granulocyte-macrophage colony-stimulating factor-induced activation of STAT5A in human monocyte-derived macrophages.

Authors:  Tammra J Warby; Suzanne M Crowe; Anthony Jaworowski
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Review 8.  Strategies for Delivery of siRNAs to Ovarian Cancer Cells.

Authors:  Rossella Farra; Matea Maruna; Francesca Perrone; Mario Grassi; Fabio Benedetti; Marianna Maddaloni; Maguie El Boustani; Salvo Parisi; Flavio Rizzolio; Giancarlo Forte; Fabrizio Zanconati; Maja Cemazar; Urska Kamensek; Barbara Dapas; Gabriele Grassi
Journal:  Pharmaceutics       Date:  2019-10-22       Impact factor: 6.321

  8 in total

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