Literature DB >> 8603420

alpha6beta1-Integrin, a major cell surface carrier of beta1-6-branched oligosaccharides, mediates migration of EJ-ras-transformed fibroblasts on laminin-1 independently of its glycosylation state.

M G Jasiulionis1, R Chammas, A M Ventura, L R Travassos, R R Brentani.   

Abstract

EJ-ras oncogene-induced malignant transformation is characterized by a series of changes in cell surface carbohydrates and cell-cell and cell-matrix interactions. Here, we show that EJ-ras-transformed NIH-3T3 fibroblasts acquired a migratory phenotype on laminin-1 surfaces. Such a phenotype was accompanied by overexpression of: (a) functional alpha6beta1, but not other laminin binding beta1-integrins; and (b) glycoconjugates on the cell surface bearing large oligosaccharides recognized by leukoagglutinin from Phaseolus vulgaris (L-PHA). The internal pool of pre-beta1-integrins was differently regulated in EJ-ras-transformed cells compared with nontransfected fibroblasts. Conversion of pre-beta1- into mature beta1-integrins was faster in EJ-ras-transformed cells, a process associated with the overexpression of the alpha6-chain. Overexpression of L-PHA-reactive oligosaccharides is dependent on the activity of N-acetylglucosaminyltransferase V, which is increased in transformed cells [J. W. Dennis et al., Science (Washington DC), 236: 582-585, 1987]. We show that beta1-integrins were the major carriers of L-PHA-reactive oligosaccharides on the cell surface. This glycosylation pattern, however, was not necessary for either the cell surface expression of beta1-integrins or their functional activity in the migratory response to laminin-1. Moreover, EJ-ras-transformed fibroblasts aggregated spontaneously. These effects were not observed in c-jun-transfected fibroblasts, which were unable to migrate on laminin, did not overexpress either beta1-integrins or L-PHA-reactive oligosaccharides, and did not self-aggregate.

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Year:  1996        PMID: 8603420

Source DB:  PubMed          Journal:  Cancer Res        ISSN: 0008-5472            Impact factor:   12.701


  6 in total

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Authors:  Xiaoyan Shen; Myoung-Soon Hong; Joel Moss; Martha Vaughan
Journal:  Proc Natl Acad Sci U S A       Date:  2007-01-16       Impact factor: 11.205

Review 2.  Regulation of N-acetylglucosaminyltransferase V and Asn-linked oligosaccharide beta(1,6) branching by a growth factor signaling pathway and effects on cell adhesion and metastatic potential.

Authors:  M Pierce; P Buckhaults; L Chen; N Fregien
Journal:  Glycoconj J       Date:  1997-08       Impact factor: 2.916

3.  Melanocyte transformation associated with substrate adhesion impediment.

Authors:  Sueli M Oba-Shinjo; Mariangela Correa; Tatiana I Ricca; Fernanda Molognoni; Maria A Pinhal; Izabel A Neves; Sueli K Marie; Lúcia O Sampaio; Helena B Nader; Roger Chammas; Miriam G Jasiulionis
Journal:  Neoplasia       Date:  2006-03       Impact factor: 5.715

Review 4.  Extracellular functions of galectin-3.

Authors:  Josiah Ochieng; Vyacheslav Furtak; Pavel Lukyanov
Journal:  Glycoconj J       Date:  2002       Impact factor: 2.916

5.  The promigratory activity of the matricellular protein galectin-3 depends on the activation of PI-3 kinase.

Authors:  Fabiana H M Melo; Diego Butera; Mara de Souza Junqueira; Daniel K Hsu; Ana Maria Moura da Silva; Fu-Tong Liu; Marinilice F Santos; Roger Chammas
Journal:  PLoS One       Date:  2011-12-28       Impact factor: 3.240

6.  Timp1 interacts with beta-1 integrin and CD63 along melanoma genesis and confers anoikis resistance by activating PI3-K signaling pathway independently of Akt phosphorylation.

Authors:  Mariana Toricelli; Fabiana H M Melo; Giovani B Peres; Débora C P Silva; Miriam G Jasiulionis
Journal:  Mol Cancer       Date:  2013-03-25       Impact factor: 27.401

  6 in total

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