Literature DB >> 8560680

Hormone and antihormone withdrawal: implications for the management of androgen-independent prostate cancer.

H I Scher1, Z F Zhang, D Nanus, W K Kelly.   

Abstract

OBJECTIVES: To analyze reported clinical outcomes for patients in whom an agent that acts via a steroid hormone receptor was "withdrawn."
METHODS: Published reports where agent(s) known to act via steroid hormone receptors were discontinued in patients with relapsing prostatic cancer were retrieved from MEDLINE listings. The trials included patients who progressed on steroidal and nonsteroidal antiandrogens, progestational agents, and estrogens. Included were the specifics of all treatments administered prior to discontinuation of the drugs, concomitant therapies, and factors that might predict a favorable response to "withdrawal."
RESULTS: Withdrawal responses were observed following the discontinuation of the antiandrogens flutamide and bicalutamide, flutamide plus aminoglutethimide, estrogens, and progestational agents. In most responding cases, responses were seen in patients with long exposure to the drug. No specific factors were predictive for response.
CONCLUSIONS: Withdrawal responses to agents that act via steroid hormone receptors represent a generalized phenomenon that can result in palliation for patients with hormonally relapsed prostate cancer. A trial of "withdrawal therapy" is warranted in patients with relapsing disease prior to the initiation of more toxic therapies. Failure to control for this phenomenon in clinical trials may lead to false attribution of response to a study agent. The data provide support for the concept that androgen independence does not necessarily mean that a tumor is resistant to further hormonal manipulations.

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Year:  1996        PMID: 8560680     DOI: 10.1016/s0090-4295(96)80011-2

Source DB:  PubMed          Journal:  Urology        ISSN: 0090-4295            Impact factor:   2.649


  6 in total

Review 1.  Prostate cancer: 11. Alternative approaches and the future of treatment.

Authors:  J Trachtenberg; J Crook; I F Tannock
Journal:  CMAJ       Date:  1999-02-23       Impact factor: 8.262

2.  Testosterone: its role in development of prostate cancer and potential risk from use as hormone replacement therapy.

Authors:  S Slater; R T Oliver
Journal:  Drugs Aging       Date:  2000-12       Impact factor: 3.923

Review 3.  Current topics and perspectives relating to hormone therapy for prostate cancer.

Authors:  Hiroyoshi Suzuki; Naoto Kamiya; Takashi Imamoto; Koji Kawamura; Masashi Yano; Makoto Takano; Takanobu Utsumi; Yukio Naya; Tomohiko Ichikawa
Journal:  Int J Clin Oncol       Date:  2008-10-23       Impact factor: 3.402

4.  Design and end points of clinical trials for patients with progressive prostate cancer and castrate levels of testosterone: recommendations of the Prostate Cancer Clinical Trials Working Group.

Authors:  Howard I Scher; Susan Halabi; Ian Tannock; Michael Morris; Cora N Sternberg; Michael A Carducci; Mario A Eisenberger; Celestia Higano; Glenn J Bubley; Robert Dreicer; Daniel Petrylak; Philip Kantoff; Ethan Basch; William Kevin Kelly; William D Figg; Eric J Small; Tomasz M Beer; George Wilding; Alison Martin; Maha Hussain
Journal:  J Clin Oncol       Date:  2008-03-01       Impact factor: 44.544

5.  Cellular androgen content influences enzalutamide agonism of F877L mutant androgen receptor.

Authors:  Daniel J Coleman; Kathryn Van Hook; Carly J King; Jacob Schwartzman; Robert Lisac; Joshua Urrutia; Archana Sehrawat; Josha Woodward; Nicholas J Wang; Roman Gulati; George V Thomas; Tomasz M Beer; Martin Gleave; James E Korkola; Lina Gao; Laura M Heiser; Joshi J Alumkal
Journal:  Oncotarget       Date:  2016-06-28

6.  Reappraisal of glucocorticoids in castrate-resistant prostate cancer.

Authors:  Oliver Sartor; Christopher C Parker; Johann de Bono
Journal:  Asian J Androl       Date:  2014 Sep-Oct       Impact factor: 3.285

  6 in total

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