Literature DB >> 8465363

Transforming growth factor-beta 1 reduces infarct size after experimental cerebral ischemia in a rabbit model.

C E Gross1, M M Bednar, D B Howard, M B Sporn.   

Abstract

BACKGROUND AND
PURPOSE: The aim of this study was to examine the effect of transforming growth factor-beta 1, a cytokine shown to amelioriate cardiac ischemia, in a rabbit model of thromboembolic stroke.
METHODS: An autologous clot embolus was introduced intracranially through the right internal carotid artery in 21 New Zealand White rabbits, with seven in each group receiving either vehicle control (albumin) or 10 or 50 micrograms transforming growth factor-beta 1 administered as an intracarotid bolus immediately before autologous clot embolization. Multiple physiological parameters were monitored, including regional cerebral blood flow, arterial blood gases, hematocrit, glucose, core temperature, and mean arterial pressure. The brain was harvested 4 hours after embolization, and infarct size was determined planimetrically as a percentage of the entire hemisphere.
RESULTS: Brain infarct size was reduced in both the 10-microgram (16.7 +/- 4.0% [mean +/- SEM], p < 0.05) and 50-microgram (21.7 +/- 4.5%) transforming growth factor-beta 1-treated groups when compared with the control group (31.9 +/- 6.6%). Regional cerebral blood flow did not show any significant intergroup or intragroup variation over time, although the 10-microgram transforming growth factor-beta 1 group experienced a greater return of cerebral blood flow in the first 2 hours after embolization.
CONCLUSIONS: Transforming growth factor-beta 1 reduced brain infarct size in a rabbit model of thromboembolic stroke. This effect was not related to a direct effect on blood flow. Studies are ongoing to determine the mechanism by which transforming growth factor-beta 1 salvages ischemic brain.

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Year:  1993        PMID: 8465363     DOI: 10.1161/01.str.24.4.558

Source DB:  PubMed          Journal:  Stroke        ISSN: 0039-2499            Impact factor:   7.914


  22 in total

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Review 4.  Trends and future developments in the pharmacological treatment of acute ischaemic stroke.

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Review 5.  A major advance in the use of growth factors to enhance wound healing.

Authors:  M B Sporn; A B Roberts
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7.  Neutrophil and Platelet Activity and Quantification Following Delayed tPA Therapy in a Rabbit Model of Thromboembolic Stroke.

Authors: 
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8.  Effect of serum on intracellular calcium homeostasis and survival of primary cortical and hippocampal CA1 neurons following brief glutamate treatment.

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Journal:  Metab Brain Dis       Date:  1994-12       Impact factor: 3.584

9.  Transforming growth factor-beta1 regulates the fate of cultured spinal cord-derived neural progenitor cells.

Authors:  S M Park; J S Jung; M S Jang; K S Kang; S K Kang
Journal:  Cell Prolif       Date:  2008-04       Impact factor: 6.831

10.  Hypoxia-inducible factor 1alpha stabilization by carbon monoxide results in cytoprotective preconditioning.

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