Literature DB >> 846014

Ozone: a possible cause of hemolytic anemia in glucose-6-phosphate dehydrogenase deficient individuals.

E J Calabrese, W H Kojola, B W Carnow.   

Abstract

A theoretical model is described that predicts that individuals with a glucose-6-phosphate dehydrogenase deficiency may experience acute hemolysis on exposure to ozone at levels reached in certain urban centers.

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Year:  1977        PMID： 846014     DOI： 10.1080/15287397709529470

Source DB:  PubMed          Journal:  J Toxicol Environ Health        ISSN： 0098-4108


  4 in total

1.  Animal model of human disease. Increased sensitivity to ozone. Animal model: mice with low levels of G-6-PD.

Authors:  E J Calabrese
Journal:  Am J Pathol       Date:  1978-05       Impact factor: 4.307

2.  Effect of in vivo ozone exposure to dorset sheep, an animal model with low levels of erythrocyte glucose-6-phosphate dehydrogenase activity.

Authors:  G S Moore; E J Calabrese; E Schulz
Journal:  Bull Environ Contam Toxicol       Date:  1981-02       Impact factor: 2.151

3.  The C57L/J mouse strain as a model for extrapulmonary effects of ozone exposure.

Authors:  G S Moore; E J Calabrese; R A Grinberg-Funes
Journal:  Bull Environ Contam Toxicol       Date:  1980-10       Impact factor: 2.151

4.  Effects of environmental oxidant stressors on individuals with a G-6-PD deficiency with particular reference to an animal model.

Authors:  E J Calabrese; G Moore; R Brown
Journal:  Environ Health Perspect       Date:  1979-04       Impact factor: 9.031
  4 in total

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