Literature DB >> 8376975

Trans-activation of the long terminal repeat of human immunodeficiency virus type 1 by the parvovirus B19 NS1 gene product.

N Sol1, F Morinet, M Alizon, U Hazan.   

Abstract

Persistent parvovirus B19 infections in human immunodeficiency virus type 1 (HIV-1)-infected patients have been reported. The two viruses could share common target cells. The NS1 protein of B19 regulates B19 expression and we have investigated its possible effect on the long terminal repeat (LTR) of HIV-1. In transient transfection experiments, NS1 trans-activated the expression of reporter genes under the control of the HIV-1 LTR. The effect of NS1 was apparent only in the presence of the HIV-1 Tat protein, and required intact TAR and TATA box sequences.

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Year:  1993        PMID： 8376975     DOI： 10.1099/0022-1317-74-9-2011

Source DB:  PubMed          Journal:  J Gen Virol        ISSN： 0022-1317            Impact factor:   3.891


  12 in total

1.  The 3' untranslated region of the B19 parvovirus capsid protein mRNAs inhibits its own mRNA translation in nonpermissive cells.

Authors:  C Pallier; A Greco; J Le Junter; A Saib; I Vassias; F Morinet
Journal:  J Virol       Date:  1997-12       Impact factor: 5.103

2.  Regulation of tumor necrosis factor alpha promoter by human parvovirus B19 NS1 through activation of AP-1 and AP-2.

Authors:  Yi Fu; Keiko Kumura Ishii; Yasuhiko Munakata; Takako Saitoh; Mitsuo Kaku; Takeshi Sasaki
Journal:  J Virol       Date:  2002-06       Impact factor: 5.103

3.  A cytotoxic nonstructural protein, NS1, of human parvovirus B19 induces activation of interleukin-6 gene expression.

Authors:  S Moffatt; N Tanaka; K Tada; M Nose; M Nakamura; O Muraoka; T Hirano; K Sugamura
Journal:  J Virol       Date:  1996-12       Impact factor: 5.103

4.  Parvovirus infection-induced cell death and cell cycle arrest.

Authors:  Aaron Yun Chen; Jianming Qiu
Journal:  Future Virol       Date:  2010-11       Impact factor: 1.831

5.  Possible interactions between the NS-1 protein and tumor necrosis factor alpha pathways in erythroid cell apoptosis induced by human parvovirus B19.

Authors:  N Sol; J Le Junter; I Vassias; J M Freyssinier; A Thomas; A F Prigent; B B Rudkin; S Fichelson; F Morinet
Journal:  J Virol       Date:  1999-10       Impact factor: 5.103

6.  Characterization of cis-acting and NS1 protein-responsive elements in the p6 promoter of parvovirus B19.

Authors:  R Gareus; A Gigler; A Hemauer; M Leruez-Ville; F Morinet; H Wolf; S Modrow
Journal:  J Virol       Date:  1998-01       Impact factor: 5.103

7.  Spontaneous mutations in the env gene of the human immunodeficiency virus type 1 NDK isolate are associated with a CD4-independent entry phenotype.

Authors:  J Dumonceaux; S Nisole; C Chanel; L Quivet; A Amara; F Baleux; P Briand; U Hazan
Journal:  J Virol       Date:  1998-01       Impact factor: 5.103

8.  Human parvovirus B19 nonstructural (NS1) protein induces apoptosis in erythroid lineage cells.

Authors:  S Moffatt; N Yaegashi; K Tada; N Tanaka; K Sugamura
Journal:  J Virol       Date:  1998-04       Impact factor: 5.103

Review 9.  Parvovirus B19 infection.

Authors:  J R Kerr
Journal:  Eur J Clin Microbiol Infect Dis       Date:  1996-01       Impact factor: 3.267

10.  Human parvovirus B19 NS1 protein modulates inflammatory signaling by activation of STAT3/PIAS3 in human endothelial cells.

Authors:  Anja Duechting; Carsten Tschöpe; Heike Kaiser; Tobias Lamkemeyer; Nobuyuki Tanaka; Susanne Aberle; Florian Lang; Joseph Torresi; Reinhard Kandolf; C-Thomas Bock
Journal:  J Virol       Date:  2008-06-11       Impact factor: 5.103
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