Literature DB >> 8249571

Arguments for a vascular glaucoma etiology.

B Sonnsjö1, C E Krakau.   

Abstract

A material of Open Angle Glaucoma (G) collected by an eye practitioner (B S) from 1980 to 1991 is analysed with special respect to the occurrence of disc haemorrhages (Hs) and retinal vein occlusions (VOs). There is a close association between Hs and VOs and G, which in all categories increases with increasing follow-up time. Furthermore, the occurrence of mixed cases--H in one eye and VO in the same or the other--support the opinion that Hs, branch vein occlusions and central vein occlusions are manifestations of the same vascular disease, the difference between them being the size of the vessel affected. The well known morphological changes in the retinal veins of glaucomas and in central vein occlusions are endothelial proliferations causing progressive increase of flow resistance. Similar changes most probably also cause branch occlusions and disc haemorrhages. The vascular affection which obstructs the vessels with hindrance of the blood flow and impaired nutrition of neuronal tissue, might be the primary cause of glaucoma.

Entities:  

Mesh:

Year:  1993        PMID: 8249571     DOI: 10.1111/j.1755-3768.1993.tb04615.x

Source DB:  PubMed          Journal:  Acta Ophthalmol (Copenh)        ISSN: 0001-639X


  11 in total

1.  Optic nerve compression by normal carotid artery in patients with normal tension glaucoma.

Authors:  N Ogata; M Imaizumi; H Kurokawa; M Arichi; M Matsumura
Journal:  Br J Ophthalmol       Date:  2005-02       Impact factor: 4.638

2.  The impact of disc hemorrhage studies on our understanding of glaucoma: a systematic review 50 years after the rediscovery of disc hemorrhage.

Authors:  Tetsuya Yamamoto
Journal:  Jpn J Ophthalmol       Date:  2018-11-21       Impact factor: 2.447

3.  Plasma vascular endothelial growth factor, soluble VEGF receptor FLT-1, and von Willebrand factor in glaucoma.

Authors:  P L Lip; D C Felmeden; A D Blann; N Matheou; S Thakur; I A Cunliffe; G Y H Lip
Journal:  Br J Ophthalmol       Date:  2002-11       Impact factor: 4.638

4.  Recurrent disc hemorrhage does not increase the rate of visual field progression.

Authors:  Heather C de Beaufort; Carlos Gustavo V De Moraes; Christopher C Teng; Tiago S Prata; Celso Tello; Robert Ritch; Jeffrey M Liebmann
Journal:  Graefes Arch Clin Exp Ophthalmol       Date:  2010-02-25       Impact factor: 3.117

5.  Hypercapnia invokes an acute loss of contrast sensitivity in untreated glaucoma patients.

Authors:  S L Hosking; D W Evans; S J Embleton; B Houde; J F Amos; J D Bartlett
Journal:  Br J Ophthalmol       Date:  2001-11       Impact factor: 4.638

6.  Ocular haemodynamic responses to induced hypercapnia and hyperoxia in glaucoma.

Authors:  S L Hosking; A Harris; H S Chung; C P Jonescu-Cuypers; L Kagemann; E J Roff Hilton; H Garzozi
Journal:  Br J Ophthalmol       Date:  2004-03       Impact factor: 4.638

7.  Structural glaucomatous progression before and after occurrence of an optic disc haemorrhage.

Authors:  Esther Chung; Anna M Demetriades; Paul J Christos; Nathan M Radcliffe
Journal:  Br J Ophthalmol       Date:  2014-07-23       Impact factor: 4.638

8.  Disc hemorrhages in patients with both normal tension glaucoma and branch retinal vein occlusion in different eyes.

Authors:  Young Cheol Yoo; Ki Ho Park
Journal:  Korean J Ophthalmol       Date:  2007-12

9.  Comparison of myopic and nonmyopic disc hemorrhage in primary open-angle glaucoma.

Authors:  Ho Soong Kim; Ki Ho Park; Jin Wook Jeoung; Joohyun Park
Journal:  Jpn J Ophthalmol       Date:  2012-11-27       Impact factor: 2.447

10.  Morphology of optic disc through heidelberg retina tomograph in retinal vein occlusions alone or in combination with primary open angle glaucoma.

Authors:  Alessandro Guido Actis; Luca Belli; Laura Dall'orto; Rachele Penna; Beatrice Brogliatti; Teresa Rolle
Journal:  Open Ophthalmol J       Date:  2013-07-26
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