Literature DB >> 8240109

Variable retinal and neurologic manifestations in patients harboring the mitochondrial DNA 8993 mutation.

R G Ortiz1, N J Newman, J M Shoffner, A E Kaufman, D A Koontz, D C Wallace.   

Abstract

OBJECTIVE: Ophthalmologic and neurologic manifestations of the mitochondrial DNA mutation at position 8993 (MTATP*NARP8993) are reported and compared with previously published reports of patients with the 8993 mutation and other mitochondrial disorders.
DESIGN: Pedigree analysis.
SETTING: University referral center. PATIENTS: Eight subjects from two unrelated pedigrees that were positive for the mitochondrial DNA replacement mutation at nucleotide position 8993 were evaluated ophthalmologically and neurologically.
RESULTS: Retinal abnormalities ranged from mild salt-and-pepper changes to severe retinitis pigmentosa-like changes with maculopathy. Neurologic manifestations were also highly variable and ranged from migraine headaches to severe dementia and Leigh's disease.
CONCLUSIONS: The type and extent of retinal pigmentary changes and neurologic findings varied substantially, even among members of the same family. These changes, although not specific for the MTATP*NARP8993 mutation, are highly suggestive of mitochondrial disease.

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Year:  1993        PMID: 8240109     DOI: 10.1001/archopht.1993.01090110091031

Source DB:  PubMed          Journal:  Arch Ophthalmol        ISSN: 0003-9950


  19 in total

1.  Genetic counseling and prenatal diagnosis for the mitochondrial DNA mutations at nucleotide 8993.

Authors:  S L White; V R Collins; R Wolfe; M A Cleary; S Shanske; S DiMauro; H H Dahl; D R Thorburn
Journal:  Am J Hum Genet       Date:  1999-08       Impact factor: 11.025

2.  Mitochondrial DNA mutations at nucleotide 8993 show a lack of tissue- or age-related variation.

Authors:  S L White; S Shanske; J J McGill; H Mountain; M T Geraghty; S DiMauro; H H Dahl; D R Thorburn
Journal:  J Inherit Metab Dis       Date:  1999-12       Impact factor: 4.982

Review 3.  Transport ATPases in biological systems and relationship to human disease: a brief overview.

Authors:  Peter L Pedersen
Journal:  J Bioenerg Biomembr       Date:  2002-10       Impact factor: 2.945

Review 4.  Mitochondrial DNA genetics and the heteroplasmy conundrum in evolution and disease.

Authors:  Douglas C Wallace; Dimitra Chalkia
Journal:  Cold Spring Harb Perspect Biol       Date:  2013-11-01       Impact factor: 10.005

Review 5.  The neuro-ophthalmology of mitochondrial disease.

Authors:  J Alexander Fraser; Valérie Biousse; Nancy J Newman
Journal:  Surv Ophthalmol       Date:  2010-05-14       Impact factor: 6.048

Review 6.  Mitochondria as chi.

Authors:  Douglas C Wallace
Journal:  Genetics       Date:  2008-06       Impact factor: 4.562

7.  De novo mutation in the mitochondrial ATP synthase subunit 6 gene (T8993G) with rapid segregation resulting in Leigh syndrome in the offspring.

Authors:  M H Tulinius; M Houshmand; N G Larsson; E Holme; A Oldfors; E Holmberg; J Wahlström
Journal:  Hum Genet       Date:  1995-09       Impact factor: 4.132

8.  Calcium homeostasis and reactive oxygen species production in cells transformed by mitochondria from individuals with sporadic Alzheimer's disease.

Authors:  J P Sheehan; R H Swerdlow; S W Miller; R E Davis; J K Parks; W D Parker; J B Tuttle
Journal:  J Neurosci       Date:  1997-06-15       Impact factor: 6.167

9.  Adaptive optics scanning laser ophthalmoscopy images in a family with the mitochondrial DNA T8993C mutation.

Authors:  Michael K Yoon; Austin Roorda; Yuhua Zhang; Chiaki Nakanishi; Lee-Jun C Wong; Qing Zhang; Leslie Gillum; Ari Green; Jacque L Duncan
Journal:  Invest Ophthalmol Vis Sci       Date:  2008-11-07       Impact factor: 4.799

10.  Controversies in counseling for mitochondrial conditions.

Authors:  A Kupelian; R E Falk; J Klein; P Fournier; N Fischel-Ghodsian
Journal:  J Genet Couns       Date:  1996-03       Impact factor: 2.537

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