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Literature DB >> 8210181

Molecular basis for Ca2+ channel diversity.

F Hofmann¹, M Biel, V Flockerzi.

Abstract

Mesh：

Substances：
Calcium Channels

Year: 1994 PMID： 8210181 DOI： 10.1146/annurev.ne.17.030194.002151

Source DB: PubMed Journal: Annu Rev Neurosci ISSN： 0147-006X Impact factor: 12.449

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114 in total

1. Pregabalin may represent a novel class of anxiolytic agents with a broad spectrum of activity.

Authors: M J Field; R J Oles; L Singh
Journal: Br J Pharmacol Date: 2001-01 Impact factor: 8.739

2. Involvement of both G protein alphas and beta gamma subunits in beta-adrenergic stimulation of vascular L-type Ca(2+) channels.

Authors: P Viard; N Macrez; C Mironneau; J Mironneau
Journal: Br J Pharmacol Date: 2001-02 Impact factor: 8.739

3. The amino side of the C-terminus determines fast inactivation of the T-type calcium channel alpha1G.

Authors: M Staes; K Talavera; N Klugbauer; J Prenen; L Lacinova; G Droogmans; F Hofmann; B Nilius
Journal: J Physiol Date: 2001-01-01 Impact factor: 5.182

4. Allosteric modulation of Ca2+ channels by G proteins, voltage-dependent facilitation, protein kinase C, and Ca(v)beta subunits.

Authors: S Herlitze; H Zhong; T Scheuer; W A Catterall
Journal: Proc Natl Acad Sci U S A Date: 2001-04-10 Impact factor: 11.205

5. Functional expression of the L-type calcium channel in mice skeletal muscle during prenatal myogenesis.

Authors: C Strube; Y Tourneur; C Ojeda
Journal: Biophys J Date: 2000-03 Impact factor: 4.033

6. The alpha1B Ca2+ channel amino terminus contributes determinants for beta subunit-mediated voltage-dependent inactivation properties.

Authors: G J Stephens; K M Page; Y Bogdanov; A C Dolphin
Journal: J Physiol Date: 2000-06-01 Impact factor: 5.182

Molecular basis for Ca2+ channel diversity.

1. Pregabalin may represent a novel class of anxiolytic agents with a broad spectrum of activity.

2. Involvement of both G protein alphas and beta gamma subunits in beta-adrenergic stimulation of vascular L-type Ca(2+) channels.

3. The amino side of the C-terminus determines fast inactivation of the T-type calcium channel alpha1G.

4. Allosteric modulation of Ca2+ channels by G proteins, voltage-dependent facilitation, protein kinase C, and Ca(v)beta subunits.

5. Functional expression of the L-type calcium channel in mice skeletal muscle during prenatal myogenesis.

6. The alpha1B Ca2+ channel amino terminus contributes determinants for beta subunit-mediated voltage-dependent inactivation properties.

Review 7. Parameters of calcium homeostasis in normal neuronal ageing.

Review 8. Molecular mechanisms of lead neurotoxicity.

9. Absence of modulation of the expressed calcium channel alpha1G subunit by alpha2delta subunits.

10. Molecular determinant for run-down of L-type Ca2+ channels localized in the carboxyl terminus of the 1C subunit.