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11 in total

Review 1. Clinical implications of matrix metalloproteinases.

Authors: Malay Mandal; Amritlal Mandal; Sudip Das; Tapati Chakraborti; Chakraborti Sajal
Journal: Mol Cell Biochem Date: 2003-10 Impact factor: 3.396

2. Mildly oxidized low-density lipoproteins suppress the proliferation of activated CD4+ T-lymphocytes and their interleukin 2 receptor expression in vitro.

Authors: S Caspar-Bauguil; M Saadawi; A Negre-Salvayre; M Thomsen; R Salvayre; H Benoist
Journal: Biochem J Date: 1998-03-01 Impact factor: 3.857

3. Angiotensin II promotes atherosclerotic lesions and aneurysms in apolipoprotein E-deficient mice.

Authors: A Daugherty; M W Manning; L A Cassis
Journal: J Clin Invest Date: 2000-06 Impact factor: 14.808

4. Evidence for a local immune response in atherosclerosis. CD4+ T cells infiltrate lesions of apolipoprotein-E-deficient mice.

Authors: X Zhou; S Stemme; G K Hansson
Journal: Am J Pathol Date: 1996-08 Impact factor: 4.307

5. Mildly oxidized low-density lipoproteins decrease early production of interleukin 2 and nuclear factor kappaB binding to DNA in activated T-lymphocytes.

Authors: S Caspar-Bauguil; J Tkaczuk; M J Haure; M Durand; J Alcouffe; M Thomsen; R Salvayre; H Benoist
Journal: Biochem J Date: 1999-01-15 Impact factor: 3.857

Immune mechanisms in atherosclerosis.

Review 1. Clinical implications of matrix metalloproteinases.

2. Mildly oxidized low-density lipoproteins suppress the proliferation of activated CD4+ T-lymphocytes and their interleukin 2 receptor expression in vitro.

3. Angiotensin II promotes atherosclerotic lesions and aneurysms in apolipoprotein E-deficient mice.

4. Evidence for a local immune response in atherosclerosis. CD4+ T cells infiltrate lesions of apolipoprotein-E-deficient mice.

5. Mildly oxidized low-density lipoproteins decrease early production of interleukin 2 and nuclear factor kappaB binding to DNA in activated T-lymphocytes.

6. Recent activation of the plaque immune response in coronary lesions underlying acute coronary syndromes.

7. Enhanced development of atherosclerosis in cholesterol-fed rabbits by suppression of cell-mediated immunity.

8. Active proliferation of different cell types, including lymphocytes, in human atherosclerotic plaques.

9. Hemodynamic modulation of monocytic cell adherence to vascular endothelium.

10. Evidence for apoptosis in advanced human atheroma. Colocalization with interleukin-1 beta-converting enzyme.