Literature DB >> 819612

Suppression of reaginic antibody (IgE) formation in mice by treatment with anti-mu antiserum.

D D Manning, J K Manning, N D Reed.   

Abstract

Neonatally initiated injection of anti-mu antiserum in mice has been shown to suppress the formation of reaginic antibodies in response to infection with the intestinal nematode, Nippostrongylus brasiliensis. This observation supports the hypothesis that IgE-producing cells arise from IgM-bearing precursors.

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Year:  1976        PMID： 819612      PMCID： PMC2190354          DOI： 10.1084/jem.144.1.288

Source DB:  PubMed          Journal:  J Exp Med        ISSN： 0022-1007            Impact factor:   14.307


  15 in total

1.  ROLE OF THE THYMUS IN IMMUNE REACTIONS IN RATS. IV. IMMUNOGLOBULINS AND ANTIBODY FORMATION.

Authors:  B G ARNASON; E H RELYVELD
Journal:  Int Arch Allergy Appl Immunol       Date:  1964

2.  The immune response of congenitally athymic (nude) mice to the intestinal nematode Nippostrongylus brasiliensis.

Authors:  R H Jacobson; N D Reed
Journal:  Proc Soc Exp Biol Med       Date:  1974-12

3.  Regulation of antibody response in vitro. IV. Heavy chain antigenic determinants on hapten-specific memory cells.

Authors:  T Kishimoto; K Ishizaka
Journal:  J Immunol       Date:  1972-12       Impact factor: 5.422

4.  Immunosuppression of congenitally athymic (nude) mice with heterologous anti-immunoglobulin heavy-chain antisera.

Authors:  D D Manning; J W Jutila
Journal:  Cell Immunol       Date:  1974-12       Impact factor: 4.868

5.  Recovery from anti-IG induced immunosuppression: implications for a model of Ig-secreting cell development.

Authors:  D D Manning
Journal:  J Immunol       Date:  1974-08       Impact factor: 5.422

6.  Immunosuppression in mice injected with heterologous anti-immunoglobulin antisera.

Authors:  D D Manning; J W Jutila
Journal:  J Immunol       Date:  1972-01       Impact factor: 5.422

7.  Induction of temporary IgA deficiency in mice injected with heterologous anti-immunoglobulin heavy chain antisera.

Authors:  D D Manning
Journal:  J Immunol       Date:  1972-11       Impact factor: 5.422

8.  Suppression of immunoglobulin class synthesis in mice. I. Effects of treatment with antibody to -chain.

Authors:  A R Lawton; R Asofsky; M B Hylton; M D Cooper
Journal:  J Exp Med       Date:  1972-02-01       Impact factor: 14.307

9.  Immunosuppression of mice injected with heterologous anti-immunoglobulin heavy chain antisera.

Authors:  D D Manning; J W Jutila
Journal:  J Exp Med       Date:  1972-06-01       Impact factor: 14.307

10.  Production of a runting syndrome and selective A deficiency in mice by the administration of anti-heavy chain antisera.

Authors:  R A Murgita; C A Mattioli; T B Tomasi
Journal:  J Exp Med       Date:  1973-07-01       Impact factor: 14.307
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  9 in total

1.  Induction of an auto-anti-IgE response in rats. III. Inhibition of a specific IgE response.

Authors:  J S Marshall; E B Bell
Journal:  Immunology       Date:  1989-03       Impact factor: 7.397

2.  Cyclophosphamide intensifies the acquisition of allergic contact dermatitis in mice rendered B-cell deficient by heterologous anti-IgM antisera.

Authors:  H C Maguire; L Faris; W Weidanz
Journal:  Immunology       Date:  1979-06       Impact factor: 7.397

3.  Expulsion of Nippostrongylus brasiliensis from mice lacking antibody production potential.

Authors:  R H Jacobson; N D Reed; D D Manning
Journal:  Immunology       Date:  1977-06       Impact factor: 7.397

4.  Anti-immunoglobulin E antibody treatment blocks histamine release and tissue contraction in sensitized mice.

Authors:  M Haak-Frendscho; R Saban; R L Shields; P M Jardieu
Journal:  Immunology       Date:  1998-05       Impact factor: 7.397

5.  Administration of an anti-IgE antibody inhibits CD23 expression and IgE production in vivo.

Authors:  M Haak-Frendscho; K Robbins; R Lyon; R Shields; J Hooley; M Schoenhoff; P Jardieu
Journal:  Immunology       Date:  1994-06       Impact factor: 7.397

6.  IgE antibody and resistance to infection. I. Selective suppression of the IgE antibody response in rats diminishes the resistance and the eosinophil response to Trichinella spiralis infection.

Authors:  A J Dessein; W L Parker; S L James; J R David
Journal:  J Exp Med       Date:  1981-02-01       Impact factor: 14.307

7.  IgE isotype suppression in anti-epsilon-treated mice.

Authors:  B E Bozelka; M L McCants; J E Salvaggio; S B Lehrer
Journal:  Immunology       Date:  1982-07       Impact factor: 7.397

8.  Protective immunity and eosinophilia in IgE-deficient SJA/9 mice infected with Nippostrongylus brasiliensis and Trichinella spiralis.

Authors:  N Watanabe; K Katakura; A Kobayashi; K Okumura; Z Ovary
Journal:  Proc Natl Acad Sci U S A       Date:  1988-06       Impact factor: 11.205

9.  T cell development in B cell-deficient mice. V. Stopping anti-mu treatment results in Igh-restricted expansion of the T suppressor cell repertoire concomitant with the development of normal immunoglobulin levels.

Authors:  K T Hayglass; B Benacerraf; M S Sy
Journal:  J Exp Med       Date:  1986-07-01       Impact factor: 14.307
  9 in total

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