Literature DB >> 8190477

Panstromal Schnyder corneal dystrophy. A clinical pathologic report with quantitative analysis of corneal lipid composition.

M McCarthy1, S Innis, P Dubord, V White.   

Abstract

BACKGROUND: The pathogenesis of Schnyder dystrophy is thought to be a primary abnormality of corneal lipid metabolism resulting in opacification secondary to lipid accumulation. Histochemical investigations have suggested the accumulated lipid includes unesterified cholesterol; however, quantitative analysis has not been done.
METHODS: Quantitative biochemical analysis was performed on a corneal button obtained from a patient with clinically and pathologically proven Schnyder dystrophy. Results were compared with four age-, sex-, and size-matched corneal buttons obtained from cadaveric eyes.
RESULTS: Corneal lipid accumulated in Schnyder dystrophy predominantly is composed of phospholipid, free cholesterol, and cholesterol ester. These constituents were markedly elevated in the Schnyder cornea when compared with cadaveric control corneas as follows: (1) phospholipid, 23.6 versus 4.05 mg/g; (2) unesterified cholesterol, 6.99 versus 0.52 mg/g; and (3) cholesterol ester, 3.16 versus 0.26 mg/g.
CONCLUSION: Phospholipid, unesterified cholesterol and cholesterol ester are the predominant lipids in Schnyder dystrophy. The pathogenesis may be a primary disorder of corneal lipid metabolism.

Entities:  

Mesh:

Year:  1994        PMID: 8190477

Source DB:  PubMed          Journal:  Ophthalmology        ISSN: 0161-6420            Impact factor:   12.079


  11 in total

1.  Localization of atherosclerosis susceptibility loci to chromosomes 4 and 6 using the Ldlr knockout mouse model.

Authors:  C L Welch; S Bretschger; N Latib; M Bezouevski; Y Guo; N Pleskac; C P Liang; C Barlow; H Dansky; J L Breslow; A R Tall
Journal:  Proc Natl Acad Sci U S A       Date:  2001-07-03       Impact factor: 11.205

2.  Visual morbidity in thirty-four families with Schnyder crystalline corneal dystrophy (an American Ophthalmological Society thesis).

Authors:  Jayne S Weiss
Journal:  Trans Am Ophthalmol Soc       Date:  2007

3.  Quantitative analysis of lipid deposits from Schnyder's corneal dystrophy.

Authors:  M Yamada; H Mochizuki; Y Kamata; Y Nakamura; Y Mashima
Journal:  Br J Ophthalmol       Date:  1998-04       Impact factor: 4.638

Review 4.  The IC3D classification of the corneal dystrophies.

Authors:  Jayne S Weiss; H U Møller; Walter Lisch; Shigeru Kinoshita; Anthony J Aldave; Michael W Belin; Tero Kivelä; Massimo Busin; Francis L Munier; Berthold Seitz; John Sutphin; Cecilie Bredrup; Mark J Mannis; Christopher J Rapuano; Gabriel Van Rij; Eung Kweon Kim; Gordon K Klintworth
Journal:  Cornea       Date:  2008-12       Impact factor: 2.651

5.  A mutation in the UBIAD1 gene in a Han Chinese family with Schnyder corneal dystrophy.

Authors:  Chunyu Du; Ying Li; Lili Dai; Lingmin Gong; Chengcheng Han
Journal:  Mol Vis       Date:  2011-10-15       Impact factor: 2.367

6.  The prenyltransferase UBIAD1 is the target of geranylgeraniol in degradation of HMG CoA reductase.

Authors:  Marc M Schumacher; Rania Elsabrouty; Joachim Seemann; Youngah Jo; Russell A DeBose-Boyd
Journal:  Elife       Date:  2015-03-05       Impact factor: 8.140

7.  A Mouse Model of Schnyder Corneal Dystrophy with the N100S Point Mutation.

Authors:  Fei Dong; Xueting Jin; Michelle A Boettler; Harrison Sciulli; Mones Abu-Asab; Christina Del Greco; Shurong Wang; Yueh-Chiang Hu; Maria M Campos; Shelley N Jackson; Ludovic Muller; Amina S Woods; Christian A Combs; Jianhua Zhang; Michael L Nickerson; Howard S Kruth; Jayne S Weiss; Winston W Kao
Journal:  Sci Rep       Date:  2018-07-05       Impact factor: 4.379

8.  Schnyder corneal dystrophy-associated UBIAD1 inhibits ER-associated degradation of HMG CoA reductase in mice.

Authors:  Youngah Jo; Jason S Hamilton; Seonghwan Hwang; Kristina Garland; Gennipher A Smith; Shan Su; Iris Fuentes; Sudha Neelam; Bonne M Thompson; Jeffrey G McDonald; Russell A DeBose-Boyd
Journal:  Elife       Date:  2019-02-20       Impact factor: 8.140

Review 9.  Corneal dystrophies.

Authors:  Gordon K Klintworth
Journal:  Orphanet J Rare Dis       Date:  2009-02-23       Impact factor: 4.123

10.  Mutations in the UBIAD1 gene, encoding a potential prenyltransferase, are causal for Schnyder crystalline corneal dystrophy.

Authors:  Andrew Orr; Marie-Pierre Dubé; Julien Marcadier; Haiyan Jiang; Antonio Federico; Stanley George; Christopher Seamone; David Andrews; Paul Dubord; Simon Holland; Sylvie Provost; Vanessa Mongrain; Susan Evans; Brent Higgins; Sharen Bowman; Duane Guernsey; Mark Samuels
Journal:  PLoS One       Date:  2007-08-01       Impact factor: 3.240

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.