Literature DB >> 8162405

Helicobacter pylori: microbiology of a 'slow' bacterial infection.

M J Blaser1.   

Abstract

The bacterium Helicobacter pylori lives in the gastric mucus layer of humans and induces a chronic inflammatory response that can result in both peptic ulceration and gastric neoplasms. Helicobacter pylori infection can be considered as a 'slow', adaptive and autoregulating process. The mechanisms by which this slow bacterial pathogen survives and interacts with the host immune system may provide a model for other persistent mucosal pathogens.

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Year:  1993        PMID: 8162405     DOI: 10.1016/0966-842x(93)90047-u

Source DB:  PubMed          Journal:  Trends Microbiol        ISSN: 0966-842X            Impact factor:   17.079


  69 in total

Review 1.  Naturally acquired human immune responses against Helicobacter pylori and implications for vaccine development.

Authors:  Y Zevering; L Jacob; T F Meyer
Journal:  Gut       Date:  1999-09       Impact factor: 23.059

2.  Detection of serum antibodies to CagA and VacA and of serum neutralizing activity for vacuolating cytotoxin in patients with Helicobacter pylori-induced gastritis.

Authors:  M Donati; S Moreno; E Storni; A Tucci; L Poli; C Mazzoni; O Varoli; V Sambri; A Farencena; R Cevenini
Journal:  Clin Diagn Lab Immunol       Date:  1997-07

3.  Molecular characterization of a flagellar export locus of Helicobacter pylori.

Authors:  S Porwollik; B Noonan; P W O'Toole
Journal:  Infect Immun       Date:  1999-05       Impact factor: 3.441

4.  Proinflammatory activity of a cecropin-like antibacterial peptide from Helicobacter pylori.

Authors:  J Bylund; T Christophe; F Boulay; T Nyström; A Karlsson; C Dahlgren
Journal:  Antimicrob Agents Chemother       Date:  2001-06       Impact factor: 5.191

5.  Fluorescence resonance energy transfer microscopy of the Helicobacter pylori vacuolating cytotoxin within mammalian cells.

Authors:  David C Willhite; Dan Ye; Steven R Blanke
Journal:  Infect Immun       Date:  2002-07       Impact factor: 3.441

6.  Expression of a human alpha-1,3/4-fucosyltransferase in the pit cell lineage of FVB/N mouse stomach results in production of Leb-containing glycoconjugates: a potential transgenic mouse model for studying Helicobacter pylori infection.

Authors:  P G Falk; L Bry; J Holgersson; J I Gordon
Journal:  Proc Natl Acad Sci U S A       Date:  1995-02-28       Impact factor: 11.205

7.  Relationship between gastric disease and deletion of cag pathogenicity island genes of Helicobacter pylori in gastric juice.

Authors:  Osamu Kawamura; Masami Murakami; Osamu Araki; Takuro Yamada; Sayaka Tomizawa; Yasuyuki Shimoyama; Keiko Minashi; Masaki Maeda; Motoyasu Kusano; Masatomo Mori
Journal:  Dig Dis Sci       Date:  2003-01       Impact factor: 3.199

8.  Selective increase of the permeability of polarized epithelial cell monolayers by Helicobacter pylori vacuolating toxin.

Authors:  E Papini; B Satin; N Norais; M de Bernard; J L Telford; R Rappuoli; C Montecucco
Journal:  J Clin Invest       Date:  1998-08-15       Impact factor: 14.808

9.  Cloning of the Helicobacter pylori recA gene and functional characterization of its product.

Authors:  W Schmitt; S Odenbreit; D Heuermann; R Haas
Journal:  Mol Gen Genet       Date:  1995-09-20

10.  Analysis of expression of CagA and VacA virulence factors in 43 strains of Helicobacter pylori reveals that clinical isolates can be divided into two major types and that CagA is not necessary for expression of the vacuolating cytotoxin.

Authors:  Z Xiang; S Censini; P F Bayeli; J L Telford; N Figura; R Rappuoli; A Covacci
Journal:  Infect Immun       Date:  1995-01       Impact factor: 3.441

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