| Literature DB >> 8102516 |
E Seböková1, I Klimes, R Moss, P Stolba, M M Wiersma, A Mitková.
Abstract
Our data indicate that (a) the existence of a defect in the clearance of circulating TG and persistence of muscle TG deposition in the high sucrose-fed neonatal streptozotocin diabetic rat, which (b) can only be partially corrected by raised dietary n-3 PUFA intake. (c) Skeletal muscle of STZ type II-like diabetic rats contains about 40% less glucose transporters, and (d) this situation cannot be changed by any of the dietary treatments employed. (e) These findings indicate that muscle TG accumulation may have no direct relation to glucose transport in muscle.Entities:
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Year: 1993 PMID： 8102516 DOI： 10.1111/j.1749-6632.1993.tb35710.x
Source DB: PubMed Journal: Ann N Y Acad Sci ISSN： 0077-8923 Impact factor: 5.691