| Literature DB >> 8044412 |
Abstract
Improved methods for quantification of organ-specific contributions to postprandial hyperlipidemia indicate substantial increases in the concentration and altered properties of hepatogenous VLDL, in addition to accumulation of chylomicron remnants. Recent clinical studies support the hypothesis that small chylomicron remnants characterized by apolipoprotein (apo)B48 and VLDL remnants characterized by apolipoprotein B100 are atherogenic lipoproteins.Entities:
Mesh:
Substances:
Year: 1994 PMID: 8044412 DOI: 10.1097/00041433-199404000-00006
Source DB: PubMed Journal: Curr Opin Lipidol ISSN: 0957-9672 Impact factor: 4.776