Literature DB >> 8038348

Phospholipase A2 activity in gastric juice from patients with active and H. pylori-eradicated healed duodenal ulcer.

K Berstad1, R Sjödahl, A Berstad.   

Abstract

BACKGROUND: Despite the close association between gastric Helicobacter pylori infection and duodenal ulcer, little is known about how this organism may promote mucosal damage. A possibility might be that it produces, or induces the host to release, phospholipases that metabolize the protective layer of phospholipids.
METHODS: Aimed at determination of phospholipase A2 activity and the concentration of choline-containing phospholipids in gastric juice, aspirates were collected during gastroscopy in 38 consecutive referrals with either active (n = 19) or H. pylori-eradicated healed duodenal ulcer (n = 19).
RESULTS: Gastric juice phospholipase A2 activity was 3.1 times higher in active than in healed duodenal ulcers (P = 0.03). Concurrently, the concentration of choline-containing phospholipids in the group with active ulcers was less than half compared with the healed group (P = 0.02).
CONCLUSION: The results indicate that phospholipase A2 activity and the concentration of choline-containing phospholipids in gastric juice are related to H. pylori status and duodenal ulcer disease.

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Year:  1994        PMID: 8038348     DOI: 10.1111/j.1365-2036.1994.tb00275.x

Source DB:  PubMed          Journal:  Aliment Pharmacol Ther        ISSN: 0269-2813            Impact factor:   8.171


  5 in total

1.  In vivo and in vitro studies of cytosolic phospholipase A2 expression in Helicobacter pylori infection.

Authors:  G Nardone; E L Holicky; J R Uhl; L Sabatino; S Staibano; A Rocco; V Colantuoni; B A Manzo; M Romano; G Budillon; F R Cockerill; L J Miller
Journal:  Infect Immun       Date:  2001-09       Impact factor: 3.441

2.  Influence of smoking and Helicobacter pylori on gastric phospholipids.

Authors:  J Wenner; T Gunnarsson; H Graffner; G Lindell
Journal:  Dig Dis Sci       Date:  2000-08       Impact factor: 3.199

3.  Acute effects of high-dose intragastric nicotine on mucosal defense mechanisms: an analysis of nicotine, prostaglandin E2, phospholipase A2, and phospholipids.

Authors:  G Lindell; K Bukhave; I Lilja; J R Madsen; H Graffner
Journal:  Dig Dis Sci       Date:  1997-03       Impact factor: 3.199

4.  Prostaglandin analogous and antioxidant activity mediated gastroprotective action of Tabernaemontana divaricata (L.) R. Br. flower methanolic extract against chemically induced gastric ulcers in rats.

Authors:  Mohammed Safwan Ali Khan; Abdul Manan Mat Jais; Adiba Afreen
Journal:  Biomed Res Int       Date:  2013-10-29       Impact factor: 3.411

5.  Accumulation of abasic sites induces genomic instability in normal human gastric epithelial cells during Helicobacter pylori infection.

Authors:  D Kidane; D L Murphy; J B Sweasy
Journal:  Oncogenesis       Date:  2014-11-24       Impact factor: 7.485

  5 in total

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