Literature DB >> 8033977

Haptoglobin development in newborn infants from diabetic mothers.

P Borgiani1, F Gloria-Bottini, G Gerlini, N Lucarini, A Amante, E Bottini.   

Abstract

Haptoglobin (Hp) development during the neonatal period has been studied in 325 newborn infants from normal pregnancies and in 242 infants from diabetic mothers. In infants from diabetic mothers Hp development is delayed as compared to infants from normal pregnancies. This delay is associated with a change in the pattern of relationship between Hp development and the polymorphism of acid phosphatase (ACP1) (an enzyme which shows phosphotyrosine phosphatase (PTPase) activity). In infants from normal pregnancies who show ACP1 phenotypes with the highest activity, the appearance of Hp is accelerated as compared to other infants. In contrast, infants from diabetic pregnancies who have ACP1 phenotypes with the highest activity, show delayed Hp development.

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Year:  1994        PMID： 8033977     DOI： 10.1007/bf01952875

Source DB:  PubMed          Journal:  Experientia        ISSN： 0014-4754


  27 in total

1.  The 18 kDa cytosolic acid phosphatase from bovine live has phosphotyrosine phosphatase activity on the autophosphorylated epidermal growth factor receptor.

Authors:  G Ramponi; G Manao; G Camici; G Cappugi; M Ruggiero; D P Bottaro
Journal:  FEBS Lett       Date:  1989-07-03       Impact factor: 4.124

2.  Haptoglobins in the newborn. I. Full-term infants.

Authors:  A G Philip
Journal:  Biol Neonate       Date:  1971

3.  Haptoglobins in the newborn. II. Low birth weight babies.

Authors:  A G Philip
Journal:  Biol Neonate       Date:  1971

4.  Serum haptoglobin appearance during neonatal period is associated with acid phosphatase (ACP1) phenotype.

Authors:  E Bottini; E Carapella; R Scacchi; N Lucarini; F Gloria-Bottini; R Pascone; E Bonci; G Maggioni
Journal:  Early Hum Dev       Date:  1985-01       Impact factor: 2.079

5.  DNA polymorphisms in the controlling region of the human haptoglobin genes: a molecular explanation for the haptoglobin 2-1 modified phenotype.

Authors:  N Maeda
Journal:  Am J Hum Genet       Date:  1991-07       Impact factor: 11.025

6.  Rat liver low M(r) phosphotyrosine protein phosphatase isoenzymes: purification and amino acid sequences.

Authors:  G Manao; L Pazzagli; P Cirri; A Caselli; G Camici; G Cappugi; A Saeed; G Ramponi
Journal:  J Protein Chem       Date:  1992-06

7.  Phenotype dependence in the inhibition of red cell acid phosphatase (ACP) by folates.

Authors:  G F Sensabaugh; V L Golden
Journal:  Am J Hum Genet       Date:  1978-09       Impact factor: 11.025

8.  Effects of maternal diabetes on early embryogenesis. Alterations in morphogenesis produced by the ketone body, B-hydroxybutyrate.

Authors:  W E Horton; T W Sadler
Journal:  Diabetes       Date:  1983-07       Impact factor: 9.461

9.  Foetal macrosomia and erythrocyte acid phosphatase (ACP1) polymorphism in diabetic and normal pregnancy.

Authors:  F Gloria-Bottini; G Gerlini; N Lucarini; P Borgiani; M C Gori; A Amante; P Lucarelli; E Bottini
Journal:  Early Hum Dev       Date:  1988 Aug-Sep       Impact factor: 2.079

10.  Classification and diagnosis of diabetes mellitus and other categories of glucose intolerance. National Diabetes Data Group.

Authors: 
Journal:  Diabetes       Date:  1979-12       Impact factor: 9.461
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  1 in total

1.  Perinatal asphyxia may present with features of neonatal atypical hemolytic uremic syndrome.

Authors:  Valérie Biran; Sébastien Fau; Taleb Jamal; Frédérique Veinberg; Sylvain Renolleau; Francis Gold; Albert Bensman; Tim Ulinski
Journal:  Pediatr Nephrol       Date:  2007-08-03       Impact factor: 3.714
  1 in total

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