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Literature DB >> 797343

Analysis of clinical and electrophysiological findings in Jakob-Creutzfeldt disease.

Abstract

Serial EEG and EMG investigations were performed in the course of a histologically verified case of Jakob-Creutzfeldt disease. Some electrophysiologic data, including visual-evoked responses were analyzed using a computer. Correlations between clinical and EEG findings suggest that the generator of the periodic discharges may be localized in some circumscribed area of the upper brain stem, probably the thalamus. The inhibitory effect of various narcotics suggests that the pacemaker of the triphasic discharges has relations to the nonspecific activating system.

Mesh：

Substances：
Diazepam

Year: 1976 PMID： 797343 DOI： 10.1007/bf00343240

Source DB: PubMed Journal: Arch Psychiatr Nervenkr (1970)

12 in total

1. [Clinical aspects & neuropathology of subacute presenile spongiform atrophy with dyskinetic terminal stage].

Authors: H JACOB; W EICKE; H ORTHNER
Journal: Dtsch Z Nervenheilkd Date: 1958

2. Subacute spongiform encephalopathy--a subacute form of encephalopathy attributable to vascular dysfunction (spongiform cerebral atrophy).

Authors: S NEVIN; W H McMENEMEY; S BEHRMAN; D P JONES
Journal: Brain Date: 1960-12 Impact factor: 13.501

3. Rapidly progressive cerebral degeneration (subacute vascular encephalopathy) with mental disorder, focal disturbances, and myoclonic epilepsy.

Authors: D P JONES; S NEVIN
Journal: J Neurol Neurosurg Psychiatry Date: 1954-05 Impact factor: 10.154

4. [The clinical syndrome of presenile idiopathic encephalopathy ("Jakob-Creutzfeldt-syndrome") or spongiform encephalopathy (author's transl)].

Authors: R Meyendorf; C Förster
Journal: Arch Psychiatr Nervenkr (1970) Date: 1973-11-27

9. Jakob-Creutzfeldt disease. Modification of clinical and electroencephalographic activity with methylphenidate and diazepam.

Authors: F Elliott; C Gardner-Thorpe; D D Barwick; J B Foster
Journal: J Neurol Neurosurg Psychiatry Date: 1974-08 Impact factor: 10.154

10. Infection as the etiology of spongiform encephalopathy (Creutzfeldt-Jakob disease).

Authors: C J Gibbs; D C Gajdusek
Journal: Science Date: 1969-09-05 Impact factor: 47.728

1 in total

1. Temporal evolution of electroencephalographic abnormalities in Creutzfeldt-Jakob disease.

Authors: G K Schlenska; G F Walter
Journal: J Neurol Date: 1989-12 Impact factor: 4.849

1 in total

Analysis of clinical and electrophysiological findings in Jakob-Creutzfeldt disease.

1. [Clinical aspects & neuropathology of subacute presenile spongiform atrophy with dyskinetic terminal stage].

2. Subacute spongiform encephalopathy--a subacute form of encephalopathy attributable to vascular dysfunction (spongiform cerebral atrophy).

3. Rapidly progressive cerebral degeneration (subacute vascular encephalopathy) with mental disorder, focal disturbances, and myoclonic epilepsy.

4. [The clinical syndrome of presenile idiopathic encephalopathy ("Jakob-Creutzfeldt-syndrome") or spongiform encephalopathy (author's transl)].

5. The periodic events of subacute sclerosing leucoencephalitis.

6. Evolution of EEG and visual evoked response changes in Jakob-Creutzfeldt disease.

7. On the origin of diffuse spikes in Jakob-Creutzfeldt's disease.

8. [Valium in a case of spongious (McMenemey-Nevin) encephalopathy].

9. Jakob-Creutzfeldt disease. Modification of clinical and electroencephalographic activity with methylphenidate and diazepam.

10. Infection as the etiology of spongiform encephalopathy (Creutzfeldt-Jakob disease).

1. Temporal evolution of electroencephalographic abnormalities in Creutzfeldt-Jakob disease.