Literature DB >> 7964738

Cortical cytochrome oxidase activity is reduced in Alzheimer's disease.

E M Mutisya1, A C Bowling, M F Beal.   

Abstract

A defect in energy metabolism may play a role in the pathogenesis of neurodegenerative diseases, such as Alzheimer's disease. In the present study, we examined the activities of the enzymes that catalyze oxidative phosphorylation in frontal, temporal, parietal, and occipital cortex from Alzheimer's disease patients and age-matched controls. Complex I and complex II-III activities showed a small decrease in occipital cortex, but were unaffected in the other cortical areas. The most consistent change was a significant decrease of cytochrome oxidase (complex IV) activity of 25-30% in the four cortical regions examined. These results provide further evidence of a cytochrome oxidase defect in Alzheimer's disease postmortem brain tissue. A deficiency in this key energy-metabolizing enzyme could lead to a reduction in energy stores and thereby contribute to the neurodegenerative process.

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Year:  1994        PMID: 7964738     DOI: 10.1046/j.1471-4159.1994.63062179.x

Source DB:  PubMed          Journal:  J Neurochem        ISSN: 0022-3042            Impact factor:   5.372


  136 in total

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Journal:  Neurochem Res       Date:  1999-07       Impact factor: 3.996

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Review 4.  Neuronal degeneration and mitochondrial dysfunction.

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Review 5.  Role of mitochondrial homeostasis and dynamics in Alzheimer's disease.

Authors:  J Eva Selfridge; Lezi E; Jianghua Lu; Russell H Swerdlow
Journal:  Neurobiol Dis       Date:  2012-01-10       Impact factor: 5.996

6.  Functional integrity of mitochondrial genomes in human platelets and autopsied brain tissues from elderly patients with Alzheimer's disease.

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Journal:  Proc Natl Acad Sci U S A       Date:  1999-03-02       Impact factor: 11.205

Review 7.  Mitochondria, OxPhos, and neurodegeneration: cells are not just running out of gas.

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Journal:  J Clin Invest       Date:  2019-01-02       Impact factor: 14.808

8.  Cytochrome c oxidase deficiency in neurons decreases both oxidative stress and amyloid formation in a mouse model of Alzheimer's disease.

Authors:  Hirokazu Fukui; Francisca Diaz; Sofia Garcia; Carlos T Moraes
Journal:  Proc Natl Acad Sci U S A       Date:  2007-08-21       Impact factor: 11.205

Review 9.  Mitochondrial Dysfunction and Synaptic Transmission Failure in Alzheimer's Disease.

Authors:  Lan Guo; Jing Tian; Heng Du
Journal:  J Alzheimers Dis       Date:  2017       Impact factor: 4.472

10.  Decline in cytochrome c oxidase activity in rat-brain mitochondria with aging. Role of peroxidized cardiolipin and beneficial effect of melatonin.

Authors:  Giuseppe Petrosillo; Valentina De Benedictis; Francesca M Ruggiero; Giuseppe Paradies
Journal:  J Bioenerg Biomembr       Date:  2013-03-15       Impact factor: 2.945

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