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Literature DB >> 7862274

Changes in the basal ganglia and thalamus following reperfusion after complete cerebral ischaemia.

M Fujioka¹, K Okuchi, S Miyamoto, T Sakaki, K Hiramatsu, M Tominaga, Y Kamada, S Iwasaki.

Abstract

We report specific changes bilaterally in the basal ganglia and thalamus following reperfusion after complete cerebral ischaemia. A 69-year-old man, resuscitated after cardiac arrest, showed symmetrical low-density lesions in the head of the caudate nucleus and lentiform nucleus on CT. MRI revealed methaemoglobin derived from minor haemorrhage in the basal ganglia and thalamus, not evident on CT. We suggest that this haemorrhage results from diapedesis of red blood cells through the damaged capillary endothelium following reperfusion.

Entities: Disease Species

Mesh：

Substances：
Methemoglobin

Year: 1994 PMID： 7862274 DOI： 10.1007/bf00600418

Source DB: PubMed Journal: Neuroradiology ISSN： 0028-3940 Impact factor: 2.804

20 in total

1. Long-term increase in coherence between the basal ganglia and motor cortex after asphyxial cardiac arrest and resuscitation in developing rats.

Authors: Bhooma R Aravamuthan; Michael Shoykhet
Journal: Pediatr Res Date: 2015-06-17 Impact factor: 3.756

1 in total

Changes in the basal ganglia and thalamus following reperfusion after complete cerebral ischaemia.

1. Cerebral ischemia. II. The no-reflow phenomenon.

2. Ischaemic brain damage: the role of excitatory activity and of calcium entry.

3. Symmetrical columnar necrosis of the basal ganglia and brain stem in an adult following cardiac arrest.

Review 4. Neurochemical correlates to selective neuronal vulnerability.

5. Selective neuronal vulnerability: morphological and molecular characteristics.

6. Diagnosis of acute cerebral infarction: comparison of CT and MR imaging.

7. Early CT findings of global central nervous system hypoperfusion.

8. No-reflow phenomenon in the cerebral circulation of the gerbil.

9. Hypotensive brain stem necrosis or cardiac arrest encephalopathy?

10. Local cerebral blood flow in the recovery period following complete cerebral ischemia in the rat.

1. Long-term increase in coherence between the basal ganglia and motor cortex after asphyxial cardiac arrest and resuscitation in developing rats.