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23 in total

1. Single-cell screening of cytosolic [Ca(2+)] reveals cell-selective action by the Alzheimer's Aβ peptide ion channel.

Authors: Hopi Lin; Nelson J Arispe
Journal: Cell Stress Chaperones Date: 2014-11-01 Impact factor: 3.667

2. Interaction of tau protein with model lipid membranes induces tau structural compaction and membrane disruption.

Authors: Emmalee M Jones; Manish Dubey; Phillip J Camp; Briana C Vernon; Jacek Biernat; Eckhard Mandelkow; Jaroslaw Majewski; Eva Y Chi
Journal: Biochemistry Date: 2012-03-14 Impact factor: 3.162

3. Measurement of the attachment and assembly of small amyloid-β oligomers on live cell membranes at physiological concentrations using single-molecule tools.

Authors: Suman Nag; Jiji Chen; J Irudayaraj; S Maiti
Journal: Biophys J Date: 2010-09-22 Impact factor: 4.033

4. Beta-amyloid causes depletion of synaptic vesicles leading to neurotransmission failure.

Authors: Jorge Parodi; Fernando J Sepúlveda; Jorge Roa; Carlos Opazo; Nibaldo C Inestrosa; Luis G Aguayo
Journal: J Biol Chem Date: 2009-11-13 Impact factor: 5.157

5. Calcium homeostasis and reactive oxygen species production in cells transformed by mitochondria from individuals with sporadic Alzheimer's disease.

Authors: J P Sheehan; R H Swerdlow; S W Miller; R E Davis; J K Parks; W D Parker; J B Tuttle
Journal: J Neurosci Date: 1997-06-15 Impact factor: 6.167

Review 6. Disordered amyloidogenic peptides may insert into the membrane and assemble into common cyclic structural motifs.

Authors: Hyunbum Jang; Fernando Teran Arce; Srinivasan Ramachandran; Bruce L Kagan; Ratnesh Lal; Ruth Nussinov
Journal: Chem Soc Rev Date: 2014-10-07 Impact factor: 54.564

The ability of amyloid beta-protein [A beta P (1-40)] to form Ca2+ channels provides a mechanism for neuronal death in Alzheimer's disease.

1. Single-cell screening of cytosolic [Ca(2+)] reveals cell-selective action by the Alzheimer's Aβ peptide ion channel.

2. Interaction of tau protein with model lipid membranes induces tau structural compaction and membrane disruption.

3. Measurement of the attachment and assembly of small amyloid-β oligomers on live cell membranes at physiological concentrations using single-molecule tools.

4. Beta-amyloid causes depletion of synaptic vesicles leading to neurotransmission failure.

5. Calcium homeostasis and reactive oxygen species production in cells transformed by mitochondria from individuals with sporadic Alzheimer's disease.

Review 6. Disordered amyloidogenic peptides may insert into the membrane and assemble into common cyclic structural motifs.

7. Zn2+ interaction with Alzheimer amyloid beta protein calcium channels.

8. Synaptotoxicity of Alzheimer beta amyloid can be explained by its membrane perforating property.

9. Small molecule blockers of the Alzheimer Abeta calcium channel potently protect neurons from Abeta cytotoxicity.

10. Functional and structural effects of amyloid-β aggregate on Xenopus laevis oocytes.