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Literature DB >> 7826144

Silicon nephropathy and myeloperoxidase antibodies.

A Chevailler, F Carrere, G Renier, D Hurez, J F Subra, P Reboul, P Riberi, C Masson.

Abstract

Entities: Disease Gene

Mesh：

Substances：

Year: 1994 PMID： 7826144 PMCID： PMC1005465 DOI： 10.1136/ard.53.11.781-b

Source DB: PubMed Journal: Ann Rheum Dis ISSN： 0003-4967 Impact factor: 19.103

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9 in total

1. Circulating antinuclear and rheumatoid factors in coal miners. A prevalence study in Pennsylvania and West Virginia.

Authors: M Lippmann; H L Eckert; N Hahon; W K Morgan
Journal: Ann Intern Med Date: 1973-12 Impact factor: 25.391

2. [Rapidly progressive glomerulonephritis in pneumoconiotic coal miners].

Authors: M Dracon; C Noël; B Wallaert; P Dequiedt; G Lelièvre; A Tacquet
Journal: Nephrologie Date: 1990

3. Anti-myeloperoxidase antibodies and associated diseases.

Authors: A Chevailler; J F Subra; G Renier; F Carrère; D Hurez
Journal: Adv Exp Med Biol Date: 1993 Impact factor: 2.622

4. Association of autoantibodies to myeloperoxidase with different forms of vasculitis.

Authors: J W Tervaert; R Goldschmeding; J D Elema; P C Limburg; M van der Giessen; M G Huitema; M I Koolen; R J Hené; T H The; G K van der Hem
Journal: Arthritis Rheum Date: 1990-08

5. Association between silica exposure and necrotizing crescentic glomerulonephritis with p-ANCA and anti-MPO antibodies: a hospital-based case-control study.

Authors: G Gregorini; A Ferioli; F Donato; P Tira; L Morassi; R Tardanico; L Lancini; R Maiorca
Journal: Adv Exp Med Biol Date: 1993 Impact factor: 2.622

6. Silica-stimulated monocytes release fibroblast proliferation factors identical to interleukin 1. A potential role for interleukin 1 in the pathogenesis of silicosis.

Authors: J A Schmidt; C N Oliver; J L Lepe-Zuniga; I Green; I Gery
Journal: J Clin Invest Date: 1984-05 Impact factor: 14.808

7. Multiple clinical and biological autoimmune manifestations in 50 workers after occupational exposure to silica.

Authors: J Sanchez-Roman; I Wichmann; J Salaberri; J M Varela; A Nuñez-Roldan
Journal: Ann Rheum Dis Date: 1993-07 Impact factor: 19.103

8. Anti-neutrophil cytoplasmic autoantibodies with specificity for myeloperoxidase in patients with systemic vasculitis and idiopathic necrotizing and crescentic glomerulonephritis.

Authors: R J Falk; J C Jennette
Journal: N Engl J Med Date: 1988-06-23 Impact factor: 91.245

9. Antimyeloperoxidase-associated proliferative glomerulonephritis: an animal model.

Authors: E Brouwer; M G Huitema; P A Klok; H de Weerd; J W Tervaert; J J Weening; C G Kallenberg
Journal: J Exp Med Date: 1993-04-01 Impact factor: 14.307

9 in total

5 in total

Silicon nephropathy and myeloperoxidase antibodies.

1. Circulating antinuclear and rheumatoid factors in coal miners. A prevalence study in Pennsylvania and West Virginia.

2. [Rapidly progressive glomerulonephritis in pneumoconiotic coal miners].

3. Anti-myeloperoxidase antibodies and associated diseases.

4. Association of autoantibodies to myeloperoxidase with different forms of vasculitis.

5. Association between silica exposure and necrotizing crescentic glomerulonephritis with p-ANCA and anti-MPO antibodies: a hospital-based case-control study.

6. Silica-stimulated monocytes release fibroblast proliferation factors identical to interleukin 1. A potential role for interleukin 1 in the pathogenesis of silicosis.

7. Multiple clinical and biological autoimmune manifestations in 50 workers after occupational exposure to silica.

8. Anti-neutrophil cytoplasmic autoantibodies with specificity for myeloperoxidase in patients with systemic vasculitis and idiopathic necrotizing and crescentic glomerulonephritis.

9. Antimyeloperoxidase-associated proliferative glomerulonephritis: an animal model.

Review 1. ANCA-associated diseases and silica exposure.

2. Microscopic polyangiitis after silicone breast implantation.

3. Lymphopenia in occupational pulmonary silicosis with or without autoimmune disease.

4. Antimyeloperoxidase antibodies in individuals with occupational exposure to silica.

Review 5. Antineutrophil cytoplasmic autoantibodies: how should the biologist manage them?