Literature DB >> 7780565

Monocular elevation paresis caused by an oculomotor fascicular impairment.

C D Gauntt1, S Kashii, I Nagata.   

Abstract

A 54-year-old man developed an acquired monocular elevation paresis. Forced lid closure or oculocephalic reflexes failed to elevate the affected eye in contrast to the contralateral eye, which turned fully upward using either maneuver. A mass lesion compressing the lateral aspect of the right oculomotor nerve at the exit of the brainstem was found. The monocular elevation paresis in this patient reflects damage of the oculomotor nerve fascicles that supply the inferior oblique and superior rectus muscles. This case supports the accepted topographical fascicular arrangement of the oculomotor nerve, with the inferior oblique and superior rectus being the most lateral and caudal, and the pupilloconstrictor fibers and the inferior rectus being most medial and rostral.

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Year:  1995        PMID: 7780565

Source DB:  PubMed          Journal:  J Neuroophthalmol        ISSN: 1070-8022            Impact factor:   3.042


  3 in total

1.  Isolated vertical ophthalmoplegia caused by bilateral rostroventral midbrain infarction.

Authors:  Hiroya Naruse; Yu Nagashima; Risa Maekawa; Yasushi Shiio
Journal:  J Neurol       Date:  2012-01-31       Impact factor: 4.849

2.  Abduction paresis with rostral pontine and/or mesencephalic lesions: Pseudoabducens palsy and its relation to the so-called posterior internuclear ophthalmoplegia of Lutz.

Authors:  F Thömke; H C Hopf
Journal:  BMC Neurol       Date:  2001-12-18       Impact factor: 2.474

Review 3.  Clinical Approach to Supranuclear Brainstem Saccadic Gaze Palsies.

Authors:  Alexandra Lloyd-Smith Sequeira; John-Ross Rizzo; Janet C Rucker
Journal:  Front Neurol       Date:  2017-08-23       Impact factor: 4.003

  3 in total

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