Literature DB >> 7743232

Clonal hematologic disorders in Down syndrome. A review.

H Avet-Loiseau1, F Mechinaud, J L Harousseau.   

Abstract

PURPOSE: Children with Down syndrome (DS) carry an elevated risk of developing clonal hematologic disorders. In this review, we summarize reported data describing the incidence of leukemia in children with DS and associated leukemic cell phenotypic and functional features. PATIENTS AND METHODS: Major leukemic subtypes, acute lymphoblastic leukemia (ALL), acute nonlymphoblastic leukemia (ANLL), myelodysplastic syndromes (MDS), and transient abnormal myelopoiesis (TAM) are evaluated in the context of patient age and treatment responsiveness.
RESULTS: It is apparent that although leukemia in children with DS is a relatively frequent event, prognosis with current conventional therapeutic strategies is excellent.
CONCLUSIONS: Treatment options for TAM, a monoclonal cellular proliferation in neonates, are discussed.

Entities:  

Mesh:

Year:  1995        PMID: 7743232     DOI: 10.1097/00043426-199502000-00003

Source DB:  PubMed          Journal:  J Pediatr Hematol Oncol        ISSN: 1077-4114            Impact factor:   1.289


  4 in total

1.  Malignancies in Down syndrome.

Authors:  P Kusumakumary; T S Vats; R Ankathil; H R Gattamaneni; M K Nair
Journal:  Indian J Pediatr       Date:  1997 Nov-Dec       Impact factor: 1.967

2.  Life-threatening complications of transient abnormal myelopoiesis in neonates with Down syndrome.

Authors:  Sabine Dormann; Marcus Krüger; Roland Hentschel; Regina Rasenack; Brigitte Strahm; Udo Kontny; Charlotte Niemeyer
Journal:  Eur J Pediatr       Date:  2004-04-28       Impact factor: 3.183

3.  Lineage conversion from acute lymphoblastic leukemia to acute myeloid leukemia on rearrangement of the IgH gene in a patient with Down syndrome.

Authors:  Kazuya Tsuboi; Makoto Yazaki; Hiroshi Miwa; Shinsuke Iida; Shogo Banno; Atsushi Wakita; Masakazu Nitta; Ryuzo Ueda
Journal:  Int J Hematol       Date:  2002-07       Impact factor: 2.490

4.  Developmental differences in IFN signaling affect GATA1s-induced megakaryocyte hyperproliferation.

Authors:  Andrew J Woo; Karen Wieland; Hui Huang; Thomas E Akie; Taylor Piers; Jonghwan Kim; Alan B Cantor
Journal:  J Clin Invest       Date:  2013-07-01       Impact factor: 14.808

  4 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.