Literature DB >> 7737652

Mitochondrial oxidative phosphorylation and intracellular glutathione compartmentation during rat liver regeneration.

G Vendemiale1, F Guerrieri, I Grattagliano, D Didonna, L Muolo, E Altomare.   

Abstract

The rate of mitochondrial oxidative phosphorylation and the cytosolic and mitochondrial total and oxidized glutathione concentrations were studied in regenerating rat livers after partial (70%) hepatectomy. The rate of mitochondrial oxidative phosphorylation progressively decreased during the early prereplicative phase of liver regeneration. This was accompanied by a progressive decrease in mitochondrial, but not cytosolic, glutathione concentration. Twenty-four hours after partial hepatectomy, both the rate of adenosine triphosphate (ATP) synthesis and the amount of mitochondrial glutathione were depressed by 50% with respect to controls (sham-operated animals). During the second replicative phase, both the oxidative phosphorylation rate and mitochondrial glutathione concentration were recovered; however, the kinetics of the recovery were different, being the total amount of mitochondrial glutathione completely restored 48 hours after partial hepatectomy, whereas 72 hours were needed for the recovery of oxidative phosphorylation. The decrease in the rate of oxidative phosphorylation, during the early phase of liver regeneration, appeared to be secondary to the decreased content of the catalytic subunit beta-F1 of the ATP synthase complex, which in turn was shown to be linearly related to the decrease of intramitochondrial glutathione. These observations suggest that the two phenomena may be due to the previously reported increased free radical production during the early phase of liver regeneration. The depression of mitochondrial glutathione after partial hepatectomy may play a contributory role in structural and functional alterations of mitochondria occurring in the first retrodifferential phase of liver regeneration.

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Year:  1995        PMID: 7737652

Source DB:  PubMed          Journal:  Hepatology        ISSN: 0270-9139            Impact factor:   17.425


  6 in total

1.  Correlation between decreased expression of mitochondrial F0F1-ATP synthase and low regenerating capability of the liver after partial hepatectomy in hypothyroid rats.

Authors:  F Guerrieri; C Nicoletti; E Adorisio; G Caraccio; P Leonetti; F Zanotti; P Cantatore
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2.  Acute ethanol preexposure promotes liver regeneration after partial hepatectomy in mice by activating ALDH2.

Authors:  Xiang Ding; Juliane I Beier; Keegan J Baldauf; Jenny D Jokinen; Hai Zhong; Gavin E Arteel
Journal:  Am J Physiol Gastrointest Liver Physiol       Date:  2013-10-31       Impact factor: 4.052

3.  Linking Mitochondrial Dysfunction to Organismal and Population Health in the Context of Environmental Pollutants: Progress and Considerations for Mitochondrial Adverse Outcome Pathways.

Authors:  David A Dreier; Danielle F Mello; Joel N Meyer; Christopher J Martyniuk
Journal:  Environ Toxicol Chem       Date:  2019-08       Impact factor: 3.742

Review 4.  Metabolic syndrome, aging and involvement of oxidative stress.

Authors:  Francesca Bonomini; Luigi Fabrizio Rodella; Rita Rezzani
Journal:  Aging Dis       Date:  2015-03-10       Impact factor: 6.745

5.  Expression of Bcl-2 family during liver regeneration and identification of Bcl-x as a delayed early response gene.

Authors:  S P Tzung; N Fausto; D M Hockenbery
Journal:  Am J Pathol       Date:  1997-06       Impact factor: 4.307

Review 6.  Current concepts of mechanisms in drug-induced hepatotoxicity.

Authors:  Stefan Russmann; Gerd A Kullak-Ublick; Ignazio Grattagliano
Journal:  Curr Med Chem       Date:  2009       Impact factor: 4.530

  6 in total

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