Literature DB >> 7619234

Maternal cells in chorionic villi from placentae of normal and abnormal human pregnancies.

C A Labarrere1, W P Faulk.   

Abstract

PROBLEM: We asked if activated macrophages and CD4 positive T lymphocytes in placental chorionic villi with villitis were of maternal or fetal origin.
METHOD: We employed a double antibody immunocytochemical technique on placental sections from three normal and four abnormal pregnancies with small-for-gestational-age infants. All studied placentae were mismatched for the maternal-fetal HLA-DRw 52 antigen. Areas of immunopathology were identified by using a monoclonal antibody to a monomorphic determination on HLA-DR, and the origin of immunological cells in areas of immunopathology was identified by using a monoclonal antibody to a polymorphic determinant on HLA-DRw 52.
RESULTS: We used a double antibody technique that employed monoclonal antibodies to HLA-DR and HLA-DRw 52 antigens and placentae that were mismatched for the maternal-fetal HLA-DRw 52 antigen. We found that the vast majority of immunological cells within villi with inflammation were of maternal origin. Quantitative studies showed that between 75 and 100% of the cells in normal as well as in abnormal pregnancies were of maternal origin, and that abnormal pregnancies had a significantly higher percentage of villi with maternal cellular infiltrates.
CONCLUSION: Our data show unequivocally that cells in areas of placental immunopathology are predominantly of maternal origin, and that abnormal pregnancies are associated with significantly more villi containing immunological cells of maternal origin.

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Year:  1995        PMID: 7619234     DOI: 10.1111/j.1600-0897.1995.tb01138.x

Source DB:  PubMed          Journal:  Am J Reprod Immunol        ISSN: 1046-7408            Impact factor:   3.886


  6 in total

1.  Maternal HLA panel-reactive antibodies in early gestation positively correlate with chronic chorioamnionitis: evidence in support of the chronic nature of maternal anti-fetal rejection.

Authors:  JoonHo Lee; Roberto Romero; Yi Xu; Jung-Sun Kim; Ji Young Park; Juan Pedro Kusanovic; Tinnakorn Chaiworapongsa; Sonia S Hassan; Chong Jai Kim
Journal:  Am J Reprod Immunol       Date:  2011-09-27       Impact factor: 3.886

2.  Involvement of Hofbauer cells and maternal T cells in villitis of unknown aetiology.

Authors:  J-S Kim; R Romero; M R Kim; Y M Kim; L Friel; J Espinoza; C J Kim
Journal:  Histopathology       Date:  2008-03       Impact factor: 5.087

3.  Villitis of unknown etiology is associated with a distinct pattern of chemokine up-regulation in the feto-maternal and placental compartments: implications for conjoint maternal allograft rejection and maternal anti-fetal graft-versus-host disease.

Authors:  Mi Jeong Kim; Roberto Romero; Chong Jai Kim; Adi L Tarca; Sovantha Chhauy; Christopher LaJeunesse; Deug-Chan Lee; Sorin Draghici; Francesca Gotsch; Juan Pedro Kusanovic; Sonia S Hassan; Jung-Sun Kim
Journal:  J Immunol       Date:  2009-03-15       Impact factor: 5.422

Review 4.  B7 family molecules as regulators of the maternal immune system in pregnancy.

Authors:  Margaret G Petroff; Antoine Perchellet
Journal:  Am J Reprod Immunol       Date:  2010-04-08       Impact factor: 3.886

Review 5.  Maternal-Fetal Inflammation in the Placenta and the Developmental Origins of Health and Disease.

Authors:  Jeffery A Goldstein; Kelly Gallagher; Celeste Beck; Rajesh Kumar; Alison D Gernand
Journal:  Front Immunol       Date:  2020-11-13       Impact factor: 7.561

6.  Metalloprotease dependent release of placenta derived fractalkine.

Authors:  Monika Siwetz; Astrid Blaschitz; Julia Kremshofer; Jelena Bilic; Gernot Desoye; Berthold Huppertz; Martin Gauster
Journal:  Mediators Inflamm       Date:  2014-03-13       Impact factor: 4.711

  6 in total

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