Literature DB >> 7605857

Role of pepsin in the development of indomethacin-induced antral ulceration in the rat.

A J Gaw1, L V Williams, C F Spraggs, C C Jordan.   

Abstract

AIMS: To examine the effects of a pepsin inhibitor, pepstatin-A, a long acting H2-receptor blocker, loxtidine, exogenous pepsin and exogenous acid against indomethacin-induced antral ulceration in the rat.
RESULTS: Indomethacin (60 mg/kg s.c.) caused antral ulceration in fasted/re-fed rats over a period of 4 h. Ulceration was prevented in a dose-dependent manner by treatment with pepstatin-A (0.1-1 mg.kg hourly) or loxtidine (3 mg/kg) given orally. Acidified methylcellulose (1 mL hourly per os) enhanced damage and also prevented protection by loxtidine (3 mg/kg per os). The protection by pepstatin-A was not altered by treatment with acidified methylcellulose but was reversed by treatment with a 10-fold excess of pepsin.
CONCLUSION: These studies suggest that mucosal degradation by pepsin, rather than direct damage by luminal acid, was the major factor in the development of indomethacin-induced antral ulceration in the rat.

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Year:  1995        PMID: 7605857     DOI: 10.1111/j.1365-2036.1995.tb00366.x

Source DB:  PubMed          Journal:  Aliment Pharmacol Ther        ISSN: 0269-2813            Impact factor:   8.171


  3 in total

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Authors:  Edward O Adeyemi; Salim A Bastaki; Irwin S Chandranath; Mohammed Y Hasan; Mohammed Fahim; Abdu Adem
Journal:  World J Gastroenterol       Date:  2005-07-21       Impact factor: 5.742

Review 2.  Peptic activity and gastroduodenal mucosal damage.

Authors:  J P Raufman
Journal:  Yale J Biol Med       Date:  1996 Jan-Feb

3.  Effects of pepsin and pepstatin on reflux tonsil hypertrophy in vitro.

Authors:  Jin Hyun Kim; Si Jung Jang; Jeong Won Yun; Myeong Hee Jung; Seung Hoon Woo
Journal:  PLoS One       Date:  2018-11-08       Impact factor: 3.240

  3 in total

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